THE NEUROPSYCHIATRIC PHENOTYPE OF NZB/W LUPUS-PRONE MOUSE MODEL AT PRE-NEPHRITIC AND NEPHRITIC STAGES OF THE DISEASE: MURINE MODEL RECAPITULATES HUMAN DISEASE

被引：1

作者：

Nikolopoulos, D. ^{[1
,2
]}

Polissidis, A. ^{[2
]}

Karali, K. ^{[2
]}

Boumpas, D. ^{[1
,2
]}

机构：

[1] Natl & Kapodistrian Univ Athens, Sch Med, Dept Internal Med 4, Attikon Univ Hosp, Athens, Greece

[2] Biomed Res Fdn Acad Athens, Athina, Greece

来源：

ANNALS OF THE RHEUMATIC DISEASES | 2020年 / 79卷

关键词：

D O I：

10.1136/annrheumdis-2020-eular.1807

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

THU0223

引用

页码：334 / 335

页数：2

共 9 条

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[4] IL-21 has a pathogenic role in a lupus-prone mouse model and its blockade with IL-21R.Fc reduces disease progression
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[5] The TLR7/8 Inhibitor Enpatoran Reverses Established Kidney Disease in the Interferon-Alpha-Accelerated NZB/W Mouse Model of Lupus
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[6] MONOTHERAPY WITH FILGOTINIB, A JAK1-SELECTIVE INHIBITOR, REDUCES DISEASE SEVERITY AND ALTERS IMMUNE CELL SUBSETS IN THE NZB/W F1 MURINE MODEL OF LUPUS
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[7] Monotherapy with Filgotinib, a JAK1-Selective Inhibitor, Reduces Disease Severity and Alters Immune Cell Subsets in the NZB/W F1 Murine Model of Lupus
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[8] Anti-vascular endothelial growth factor receptor-2 antibody accelerates renal disease in the NZB/W F1 murine systemic lupus erythematosus model
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[9] The Clinical, Oral Small Molecule IRAK4 Inhibitor, GS-5718, Improves Survival and Reduces Disease Pathologies by Modulating Multiple Inflammatory Endpoints in the Murine NZB/W Model of Spontaneous Lupus
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