Atypical PKC and TGFβ receptors co-operate to phosphorylate Par6

被引：0

|

作者：

Gunaratne, Adrian ^{[1
]}

Di Guglielmo, John ^{[1
]}

机构：

[1] Univ Western Ontario, London, ON, Canada

来源：

CANCER RESEARCH | 2011年 / 71卷

关键词：

D O I：

10.1158/1538-7445.AM2011-1051

中图分类号：

R73 [肿瘤学];

学科分类号：

100214 ;

摘要：

1051

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页数：1

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[41] Co-existence of two types of [Ca2+]i-inducing protease-activated receptors (PAR-1 and PAR-2) in rat astrocytes and C6 glioma cells
Ubl, JJ
Vöhringer, C
Reiser, G
NEUROSCIENCE, 1998, 86 (02) : 597 - 609
[42] Vitamin E forms inhibit IL-13/STAT6-induced eotaxin-3 secretion by up-regulation of PAR4, an endogenous inhibitor of atypical PKC in human lung epithelial cells
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[43] Co-existence of two types of [Ca2+]1-inducing protease-activated receptors (PAR-1 and PAR-2) in rat astrocytes and C6 glioma cells. (vol 86, pg 597, 1998)
Ubl, JJ
Vohringer, C
Reiser, G
NEUROSCIENCE, 1999, 88 (01) : 337 - 337
[44] Sustained contraction of smooth muscle by PAR1 receptors is selectively mediated by RhoA/PKC/Cpi-17 pathway:: Evidence that co-activation of the RhoA/Rho Kinase/Mypt1 pathway is suppressed by PKA released from IκBα
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Murthy, Karnam S.
GASTROENTEROLOGY, 2008, 134 (04) : A250 - A250

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