Blocking of IL-23 by anti-IL-23R antibody attenuated arthritis and decreased the synovial expression of IL-17, IL-6 and IL-10 in collagen-induced arthritis

被引:0
|
作者
Park, Young-Eun [1 ]
Kim, Jeong-Hee [2 ]
Lee, Joung-Wook [1 ]
Seo, Soo-Hong [3 ]
Cho, Mi-Ra [1 ]
Back, Seung-Hoon [4 ]
Kim, Geun-Tae [5 ]
Lee, Jun-Hee [1 ]
Kim, Sung-Il [1 ]
机构
[1] Pusan Natl Univ, Pusan, South Korea
[2] Med Res Inst, Pusan, South Korea
[3] Kosin Univ, Gospel Hosp, Pusan, South Korea
[4] Busan St Marys Med Ctr, Pusan, South Korea
[5] Busan Med Ctr, Pusan, South Korea
来源
ARTHRITIS AND RHEUMATISM | 2008年 / 58卷 / 09期
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:S260 / S260
页数:1
相关论文
共 50 条
  • [21] A Transient Breach in the Epithelial Barrier Is Associated with Increased IL-17 and IL-10 Production and Reduction of IL-23
    Amendola, Antonello
    Butera, Alessia
    Sanchez, Massimo
    Boirivant, Monica
    GASTROENTEROLOGY, 2009, 136 (05) : A151 - A151
  • [22] VITAMIN D, IL-10, IL-17, AND IL-23 AS BIOMARKER FOR DISEASE ACTIVITY IN SYSTEMIC LUPUS ERYTHEMATOSUS
    Senturk, T.
    Cetin, B. Genc
    Sargin, G.
    Aydin, N.
    ANNALS OF THE RHEUMATIC DISEASES, 2016, 75 : 1081 - 1082
  • [23] Unexpected connections of the IL-23/IL-17 and IL-4/IL-13 cytokine axes in inflammatory arthritis and enthesitis
    Bridgewood, Charlie
    Newton, Darren
    Bragazzi, Nicola
    Wittmann, Miriam
    McGonagle, Dennis
    SEMINARS IN IMMUNOLOGY, 2021, 58
  • [24] Direct osteoclastogenic potential of proinflammatory cytokines-TNFα, IL-1, IL-6, IL-17 and IL-23
    Ju, Ji Hyeon
    Cho, Mi-La
    Ko, Hyuk Jae
    Kim, Hyun Ok
    Yoon, Ho-Sung
    Kim, Sung-Soo
    Seo, Soo Hong
    Lee, Sang Heon
    Park, Sung-Hwan
    Kim, Ho-Youn
    ARTHRITIS AND RHEUMATISM, 2008, 58 (09): : S669 - S670
  • [25] Synovial RANKL/OPG MRNA expression ratio and IL-17 in collagen-induced arthritis
    Lee, J.
    Baek, S.
    Kim, G.
    Kim, J.
    Kim, S.
    ANNALS OF THE RHEUMATIC DISEASES, 2006, 65 : 78 - 78
  • [26] TGF-β is necessary for induction of IL-23R and Th17 differentiation by IL-6 and IL-23
    Morishima, Noriko
    Mizoguchi, Izuru
    Takeda, Kiyoshi
    Mizuguchi, Junichiro
    Yoshimoto, Takayuki
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2009, 386 (01) : 105 - 110
  • [27] IL-23/IL-17 axis in the pathogenesis and treatment of systemic lupus erythematosus and rheumatoid arthritis
    Izati, Aziz Farah
    Wong, Kah Keng
    Maraina, Che Hussin Che
    MALAYSIAN JOURNAL OF PATHOLOGY, 2020, 42 (03) : 333 - 347
  • [28] Association of IL-23R with as genetic susceptibility and inflammation related with elevated expression of IL-23 and IL-17 in Chinese population
    Wang, X. W.
    Huang, J. X.
    Gu, J. R.
    Lin, Z. M.
    Li, C.
    Wei, Q. J.
    Liao, Z. T.
    Jiang, Y. J.
    CLINICAL AND EXPERIMENTAL RHEUMATOLOGY, 2008, 26 (04) : 722 - 722
  • [29] IL-23 Dependent and Independent Stages of Experimental Arthritis: No Clinical Effect of Therapeutic IL-23p19 Inhibition in Collagen-induced Arthritis
    Cornelissen, Ferry
    Asmawidjaja, Patrick S.
    Mus, Adriana M. C.
    Corneth, Odilia
    Kikly, Kristine
    Lubberts, Erik
    PLOS ONE, 2013, 8 (02):
  • [30] Association of IL23r with as genetic susceptibility and inflammation related with elevated expression of IL-23 and IL-17 in Chinese population
    Gu, Jieruo
    Wang, Xinwei
    Lin, Zhiming
    Huang, Jinxian
    Li, Chao
    Wei, Qiujing
    Liao, Zetao
    Jiang, Yingjuan
    ARTHRITIS AND RHEUMATISM, 2008, 58 (09): : S349 - S349