IRF4 is essential for IL-21-mediated induction, amplification and stabilization of the Th17 phenotype

被引：0
|
作者：
Huber, M. [1 ]
Bruestle, A. [1 ]
Guralnik, A. [1 ]
Mahiny, A. [1 ]
Von Loew, E. [1 ]
Lohoff, M. [1 ]
机构：
[1] Inst Med Mikrobiol & Hyg, Marburg, Germany
来源：
WIENER KLINISCHE WOCHENSCHRIFT | 2008年 / 120卷
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：3 / 3
页数：1
相关论文
共 50 条
  • [31] Inflammasome and Fas-Mediated IL-1β Contributes to Th17/Th1 Cell Induction in Pathogenic Bacterial Infection In Vivo
    Uchiyama, Ryosuke
    Yonehara, Shin
    Taniguchi, Shun'ichiro
    Ishido, Satoshi
    Ishii, Ken J.
    Tsutsui, Hiroko
    JOURNAL OF IMMUNOLOGY, 2017, 199 (03): : 1122 - 1130
  • [32] Berberine mitigates IL-21/IL-21R mediated autophagic influx in fibroblast-like synoviocytes and regulates Th17/Treg imbalance in rheumatoid arthritis
    Dinesh, Palani
    Rasool, MahaboobKhan
    APOPTOSIS, 2019, 24 (7-8) : 644 - 661
  • [33] Berberine mitigates IL-21/IL-21R mediated autophagic influx in fibroblast-like synoviocytes and regulates Th17/Treg imbalance in rheumatoid arthritis
    Palani Dinesh
    MahaboobKhan Rasool
    Apoptosis, 2019, 24 : 644 - 661
  • [34] IRF-4 is essential for the pathogenesis of the Th1 and Th2-mediated colitis: Identification of a central regulator molecule of the IL-6-mediated apoptosis resistance
    Mudter, J
    Amoussina, L
    Weigmann, B
    Duncan, G
    Wirtz, S
    Lehr, H
    Rose-John, S
    Galle, P
    Lohoff, M
    Mak, T
    Neurath, M
    MEDIZINISCHE KLINIK, 2006, 101 (04) : A30 - A30
  • [35] 外周血IRF4、Th17细胞及其相关细胞因子与梅毒血清固定形成的关联性研究
    冀敏
    中国卫生工程学, 2017, 16 (04) : 495 - 497
  • [36] IL-4 abrogates TH17 cell-mediated inflammation by selective silencing of IL-23 in antigen-presenting cells
    Guenova, Emmanuella
    Skabytska, Yuliya
    Hoetzenecker, Wolfram
    Weindl, Guenther
    Sauer, Karin
    Tham, Manuela
    Kim, Kyu-Won
    Park, Ji-Hyeon
    Seo, Ji Hae
    Ignatova, Desislava
    Cozzio, Antonio
    Levesque, Mitchell P.
    Volz, Thomas
    Koeberle, Martin
    Kaesler, Susanne
    Thomas, Peter
    Mailhammer, Reinhard
    Ghoreschi, Kamran
    Schaekel, Knut
    Amarov, Boyko
    Eichner, Martin
    Schaller, Martin
    Clark, Rachael A.
    Roecken, Martin
    Biedermann, Tilo a
    PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2015, 112 (07) : 2163 - 2168
  • [37] IL-4 abrogates TH17 cell-mediated inflammation by selective silencing of IL-23 in antigen-presenting cells
    Guenova, E.
    Skabytska, Y.
    Hoetzenecker, W.
    Volz, T.
    Koeberle, M.
    Kaesler, S.
    Thomas, P.
    Ghoreschi, K.
    Schaekel, K.
    Schaller, M.
    Roecken, M.
    Biedermann, T.
    ALLERGY, 2015, 70 : 53 - 53
  • [38] Identification of IL-18 and th17 cells in salivary glands of patients with Sjogren's syndrome, and amplification of IL-17-mediated secretion of inflammatory cytokines from salivary gland cells by IL-18
    Sakai, Azusa
    Sugawara, Yumiko
    Kuroishi, Toshinobu
    Sasano, Takashi
    Sugawara, Shunji
    JOURNAL OF IMMUNOLOGY, 2008, 181 (04): : 2898 - 2906
  • [39] Suppression of Th1 and Th17 Responses and Induction of Treg Responses by IL-18-Expressing Plasmid Gene Combined with IL-4 on Collagen-Induced Arthritis
    Dai, Qiaomei
    Li, Yang
    Yu, Haiyue
    Wang, Xiaoyan
    BIOMED RESEARCH INTERNATIONAL, 2018, 2018
  • [40] Induction of IL-22-Producing CD4+T Cells by Segmented Filamentous Bacteria Independent of Classical Th17 Cells
    Roy, Urmi
    de Oliveira, Romulo S.
    Galvez, Eric J. C.
    Gronow, Achim
    Basic, Marijana
    Perez, Laura Garcia
    Gagliani, Nicola
    Bleich, Andre
    Huber, Samuel
    Strowig, Till
    FRONTIERS IN IMMUNOLOGY, 2021, 12
12345