Transforming Growth Factor Beta Inhibition Increases Matrix Metalloproteinase-9 Activity and Enhances Elastin Degradation in a Murine Model of Kawasaki Disease

被引：0
|
作者：
Alvira, Cristina M. [1 ]
Kim, Yu-Mee [1 ]
Guignabert, Christophe [1 ]
Wang, LingLi [1 ]
Rabinovitch, Marlene [1 ]
机构：
[1] Stanford Univ, Stanford, CA 94305 USA
来源：
CIRCULATION | 2008年 / 118卷 / 18期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：S373 / S373
页数：1
相关论文
共 50 条
  • [31] Selective inhibition of matrix metalloproteinase-9 reduces clinical severity in a murine model of Multiple Sclerosis via a reduced immune response
    Onwuha-Ekpete, Lillian C.
    Tokmina-Roszyk, Dorota
    Fields, Gregg B.
    JOURNAL OF IMMUNOLOGY, 2016, 196
  • [32] Transforming growth factor-beta differently regulates urokinase type plasminogen activator and matrix metalloproteinase-9 expression in mouse macrophages; analysis of intracellular signal transduction
    Mojsilovic, Sonja S.
    Santibanez, Juan F.
    CELL BIOLOGY INTERNATIONAL, 2015, 39 (05) : 619 - 628
  • [33] Transforming growth factor-β1 induces matrix metalloproteinase-9 expression in human meningeal cells via ERK and Smad pathways
    Okamoto, Toshio
    Takahashi, Satoru
    Nakamura, Eiki
    Nagaya, Ken
    Hayashi, Tokitsugi
    Fujieda, Kenji
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2009, 383 (04) : 475 - 479
  • [34] Tenascin-C upregulates matrix metalloproteinase-9 in breast cancer cells:: Direct and synergistic effects with transforming growth factor β1
    Kalembeyi, I
    Inada, H
    Nishiura, R
    Imanaka-Yoshida, K
    Sakakura, T
    Yoshida, T
    INTERNATIONAL JOURNAL OF CANCER, 2003, 105 (01) : 53 - 60
  • [35] Expression of matrix metalloproteinase-9 and down regulation by interferons type I/II and transforming growth factor-β1 in human monocytes
    Bauvois, B
    Lesavre, P
    Nguyen, J
    JOURNAL OF LEUKOCYTE BIOLOGY, 2005, : 50 - 50
  • [36] Transforming growth factor-β1 modulates matrix metalloproteinase-9 production through the Ras/MAPK signaling pathway in transformed keratinocytes
    Santibáñez, JF
    Guerrero, J
    Quintanilla, M
    Fabra, A
    Martínez, J
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2002, 296 (02) : 267 - 273
  • [37] Genetic polymorphism in matrix metalloproteinase-9 and transforming growth factor-β1 and susceptibility to combined pulmonary fibrosis and emphysema in a Chinese population
    Xu, Ling
    Bian, Wei
    Gu, Xiao-Hua
    Shen, Ce
    KAOHSIUNG JOURNAL OF MEDICAL SCIENCES, 2017, 33 (03): : 124 - 129
  • [38] Co-stimulation of human breast cancer cells with transforming growth factor-β and tenascin-C enhances matrix metalloproteinase-9 expression and cancer cell invasion
    Ilunga, K
    Nishiura, R
    Inada, H
    El-Karef, A
    Imanaka-Yoshida, K
    Sakakura, T
    Yoshida, T
    INTERNATIONAL JOURNAL OF EXPERIMENTAL PATHOLOGY, 2004, 85 (06) : 373 - 379
  • [39] Increased metastases are associated with inflammation and matrix metalloproteinase-9 activity at incision sites in a murine model of peritoneal dissemination of colorectal cancer
    Lee, In Kyu
    VanSaun, Michael N.
    Shim, Jung Ho
    Matrisian, Lynn M.
    Gorden, D. Lee
    JOURNAL OF SURGICAL RESEARCH, 2013, 180 (02) : 252 - 259
  • [40] ANTAGONISM OF THE P75NTR RECEPTOR INCREASES THE RELEASE OF NERVE GROWTH FACTOR IN UROTHELIAL CELLS BY INHIBITING MATRIX METALLOPROTEINASE-9
    Hamouda, Aalya
    Cammisotto, Philippe
    Sirmakesyan, Stephanie
    Saragovi, Uri
    Campeau, Lysanne
    JOURNAL OF UROLOGY, 2021, 206 : E448 - E448
12345