Cellular mechanisms in the pathogenesis of interstitial lung diseases

被引：1

作者：

Singh, Priyanka ^{[1
,2
,3
,4
]}

Ali, Saba Naaz ^{[1
,2
,3
,4
]}

Zaheer, Sufian ^{[1
,2
,3
,4
]}

Singh, Mukul ^{[1
,2
,3
,4
]}

机构：

[1] VMMC, Dept Pathol, 4th Floor,Coll Bldg, New Delhi 110029, India

[2] Safdarjang Hosp, New Delhi 110029, India

[3] Vardhman Mahavir Med Coll, Dept Pathol, New Delhi 29, India

[4] Safdarjang Hosp, New Delhi 29, India

来源：

PATHOLOGY RESEARCH AND PRACTICE | 2023年 / 248卷

关键词：

Idiopathic pulmonary fibrosis; Interstitial lung disease; Pulmonary fibrosis; Myofibroblast; IDIOPATHIC PULMONARY-FIBROSIS; EPITHELIAL-MESENCHYMAL TRANSITION; RESIDENT FIBROBLASTS; STEM-CELLS; PROLIFERATION; MYOFIBROBLAST; EXPRESSION; TRANSCRIPTION; MUTATIONS; RECEPTORS;

D O I：

10.1016/j.prp.2023.154691

中图分类号：

R36 [病理学];

学科分类号：

100104 ;

摘要：

The interstitial lung diseases (ILDs) are a large, heterogeneous group of several hundred generally rare pulmonary pathologies, which show injury, inflammation and/or scarring in the lung. Although the aetiology of these disorders remains largely unknown, various cellular mechanisms have an important role in pathogenesis of fibrosis on the background of occupational, environmental and genetic factors. We have tried to provide new insights into the interactions and cellular contributions, analysing the roles of various cells in the pathogenesis of idiopathic pulmonary fibrosis.

引用

页数：6

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