ACBP orchestrates the metabolic phenotype in Cushing's syndrome

被引：0

作者：

Paul, Mhairi ^{[1
]}

Nixon, Mark ^{[1
]}

机构：

[1] Univ Edinburgh, Univ BHF Ctr Cardiovasc Sci, Edinburgh, Scotland

来源：

NATURE METABOLISM | 2024年 / 6卷 / 12期

关键词：

LIVER;

D O I：

10.1038/s42255-024-01169-7

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

Cushing's syndrome, a condition of chronic glucocorticoid excess, disrupts metabolic homeostasis, driving fat redistribution and promoting insulin resistance. New research uses a series of elegant approaches to reveal acyl-CoA-binding protein (ACBP) as a mediator of the metabolic disturbances associated with elevated glucocorticoid levels in mice.

引用

页码：2220 / 2221

页数：2

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