ACBP orchestrates the metabolic phenotype in Cushing's syndrome

被引:0
|
作者
Paul, Mhairi [1 ]
Nixon, Mark [1 ]
机构
[1] Univ Edinburgh, Univ BHF Ctr Cardiovasc Sci, Edinburgh, Scotland
关键词
LIVER;
D O I
10.1038/s42255-024-01169-7
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
Cushing's syndrome, a condition of chronic glucocorticoid excess, disrupts metabolic homeostasis, driving fat redistribution and promoting insulin resistance. New research uses a series of elegant approaches to reveal acyl-CoA-binding protein (ACBP) as a mediator of the metabolic disturbances associated with elevated glucocorticoid levels in mice.
引用
收藏
页码:2220 / 2221
页数:2
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