The effects of grounding (earthing) on inflammation, the immune response, wound healing, and prevention and treatment of chronic inflammatory and autoimmune diseases

被引:31
|
作者
Oschman, James L. [1 ]
Chevalier, Gaetan [2 ]
Brown, Richard [3 ]
机构
[1] Nat Own Res Assoc, Dover, NH USA
[2] Univ Calif Irvine, Dev & Cell Biol Dept, 2103 McGaugh Hall, Irvine, CA 92697 USA
[3] Univ Oregon, Human Physiol Dept, Eugene, OR 97403 USA
来源
关键词
chronic inflammation; immune system; wound repair; white blood cells; macrophages; autoimmune disorders;
D O I
10.2147/JIR.S69656
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
Multi-disciplinary research has revealed that electrically conductive contact of the human body with the surface of the Earth (grounding or earthing) produces intriguing effects on physiology and health. Such effects relate to inflammation, immune responses, wound healing, and prevention and treatment of chronic inflammatory and autoimmune diseases. The purpose of this report is two-fold: to 1) inform researchers about what appears to be a new perspective to the study of inflammation, and 2) alert researchers that the length of time and degree (resistance to ground) of grounding of experimental animals is an important but usually overlooked factor that can influence outcomes of studies of inflammation, wound healing, and tumorigenesis. Specifically, grounding an organism produces measurable differences in the concentrations of white blood cells, cytokines, and other molecules involved in the inflammatory response. We present several hypotheses to explain observed effects, based on current research results and our understanding of the electronic aspects of cell and tissue physiology, cell biology, biophysics, and biochemistry. An experimental injury to muscles, known as delayed onset muscle soreness, has been used to monitor the immune response under grounded versus ungrounded conditions. Grounding reduces pain and alters the numbers of circulating neutrophils and lymphocytes, and also affects various circulating chemical factors related to inflammation.
引用
收藏
页码:83 / 96
页数:14
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