共 50 条
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- [3] TUMOR-NECROSIS-FACTOR ACTIVITY OF PULMONARY ALVEOLAR MACROPHAGES IS INCREASED BY EXPOSING LUNGS TO ENDOTOXIN OR N-O-NITRO-L ARGININE (L-NNA) [J]. FASEB JOURNAL, 1995, 9 (03): : A571 - A571
- [4] HISTAMINE-RECEPTOR BLOCKADE ATTENUATES TUMOR-NECROSIS-FACTOR ACTIVITY OF PULMONARY ALVEOLAR MACROPHAGES [J]. FASEB JOURNAL, 1991, 5 (06): : A1602 - A1602
- [5] DIPHENHYDRAMINE INCREASES TUMOR-NECROSIS-FACTOR ACTIVITY OF PULMONARY MACROPHAGES [J]. FASEB JOURNAL, 1993, 7 (03): : A368 - A368
- [6] SEPSIS INDUCES ALTERATIONS IN TUMOR-NECROSIS-FACTOR ACTIVITY OF PULMONARY MACROPHAGES [J]. FASEB JOURNAL, 1992, 6 (05): : A1611 - A1611
- [7] TUMOR-NECROSIS-FACTOR PRODUCTION BY HUMAN SARCOID ALVEOLAR MACROPHAGES [J]. AMERICAN JOURNAL OF PATHOLOGY, 1986, 125 (03): : 421 - 425
- [8] HEAT-STRESS DECREASES TUMOR-NECROSIS-FACTOR RELEASE IN LPS-STIMULATED ALVEOLAR MACROPHAGES [J]. AMERICAN REVIEW OF RESPIRATORY DISEASE, 1993, 147 (04): : A229 - A229
- [9] TUMOR NECROSIS FACTOR RELEASE BY PULMONARY ALVEOLAR MACROPHAGES [J]. THORAX, 1988, 43 (03) : P246 - P247
- [10] TUMOR-NECROSIS-FACTOR DECREASES FLUID CLEARANCE FROM ISOLATED RABBIT LUNGS [J]. FASEB JOURNAL, 1995, 9 (03): : A569 - A569