Deficiency of PI3-Kinase catalytic isoforms p110γ and p110δ in mice enhances the IL-17/G-CSF axis and induces neutrophilia

被引：0
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作者：
Kirsten Bucher
Fee Schmitt
Benedikt Mothes
Carolin Blumendeller
Daniel Schäll
Roland Piekorz
Emilio Hirsch
Bernd Nürnberg
Sandra Beer-Hammer
机构：
[1] University of Tübingen,Department of Pharmacology and Experimental Therapy, Institute of Experimental and Clinical Pharmacology and Toxicology and ICePhA mouse clinic
[2] University of Düsseldorf,Institute of Biochemistry and Molecular Biology II
[3] University of Torino,Department of Genetics, Biology and Biochemistry
[4] University Tübingen,Institute of Experimental and Clinical Pharmacology and Toxicology
来源：
Cell Communication and Signaling | / 15卷
关键词：
PI3K p110γ; PI3K p110δ,neutrophil homeostasis; IL-17-producing T cells; Il-17; G-CSF;
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