Diesel exhaust particles induce CYP1A1 and pro-inflammatory responses via differential pathways in human bronchial epithelial cells

被引：0
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作者：
Annike I Totlandsdal
Flemming R Cassee
Per Schwarze
Magne Refsnes
Marit Låg
机构：
[1] Norwegian Institute of Public Health,Department of Air Pollution and Noise, Division of Environmental Medicine
[2] National Institute for Public Health and the Environment,undefined
来源：
Particle and Fibre Toxicology | / 7卷
关键词：
Aryl Hydrocarbon Receptor; Human Bronchial Epithelial Cell; Diesel Exhaust Particle; CYP1A1 Expression; CYP1A1 mRNA;
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