Naturally occurring dominant-negative SK3 channel isoform

被引:0
|
作者
H Tomita
V G Shakkottai
G A Gutman
G Sun
W E Bunney
M D Cahalan
K G Chandy
J J Gamrgus
机构
[1] University of California Irvine,Department of Physiology and Biophysics
[2] Deaprtment of Psychiatry and Human Behavior,Department of Microbiology and Molecular Genetics
[3] University of California Irvine,Department of Pediatrics, Division of Human Genetics
[4] University of California Irvine,undefined
[5] University of California Irvine,undefined
关键词
D O I
10.1038/sj.mp.4001358
中图分类号
学科分类号
摘要
引用
收藏
页码:460 / 460
相关论文
共 50 条
  • [1] Erratum: Naturally occurring dominant-negative SK3 channel isoform
    H Tomita
    V G Shakkottai
    G A Gutman
    G Sun
    W E Bunney
    M D Cahalan
    K G Chandy
    J J Gargus
    Molecular Psychiatry, 2003, 8 : 766 - 766
  • [2] Naturally occurring dominant-negative SK3 channel isoform (vol 8, pg 460, 2003)
    Tomita, H
    Shakkottai, VG
    Gutman, GA
    Sun, G
    Bunney, WE
    Cahalan, MD
    Chandy, KG
    Gargus, JJ
    MOLECULAR PSYCHIATRY, 2003, 8 (08) : 766 - 766
  • [3] Dominant-negative truncated isoform of SK3 induces ataxia in transgenic mice
    Shakkottai, VG
    Tomita, H
    Sun, GF
    Chou, CH
    Cahalan, MD
    Gutman, G
    LaFerla, FM
    Gargus, JJ
    Chandy, KG
    FASEB JOURNAL, 2003, 17 (04): : A460 - A460
  • [4] Novel truncated isoform of SK3 potassium channel is a potent dominant-negative regulator of SK currents: implications in schizophrenia
    H Tomita
    V G Shakkottai
    G A Gutman
    G Sun
    W E Bunney
    M D Cahalan
    K G Chandy
    J J Gargus
    Molecular Psychiatry, 2003, 8 : 524 - 535
  • [5] Novel truncated isoform of SK3 potassium channel is a potent dominant-negative regulator of SK currents: implications in schizophrenia
    Tomita, H
    Shakkottai, VG
    Gutman, GA
    Sun, G
    Bunney, WE
    Cahalan, MD
    Chandy, KG
    Gargus, JJ
    MOLECULAR PSYCHIATRY, 2003, 8 (05) : 524 - 535
  • [6] Dominant-negative truncated isoform of SK3 induces ataxia in transgenic mice.
    Shakkottai, VG
    Tomita, H
    Sun, GF
    Chou, CH
    Cahalan, MD
    Gutman, G
    LaFerla, FM
    Gargus, JJ
    Chandy, KG
    BIOPHYSICAL JOURNAL, 2003, 84 (02) : 237A - 237A
  • [7] PPARγΔ5, a Naturally Occurring Dominant-Negative Splice Isoform, Impairs PPARγ Function and Adipocyte Differentiation
    Aprile, Marianna
    Cataldi, Simona
    Ambrosio, Maria Rosaria
    D'Esposito, Vittoria
    Lim, Koini
    Dietrich, Arne
    Blueher, Matthias
    Savage, David Bousfield
    Formisano, Pietro
    Ciccodicola, Alfredo
    Costa, Valerio
    CELL REPORTS, 2018, 25 (06): : 1577 - +
  • [8] A scyllatoxin-insensitive isoform of the human SK3 channel
    Wittekindt, OH
    Visan, V
    Tomita, H
    Imtiaz, F
    Gargus, JJ
    Lehmann-Horn, F
    Grissmer, S
    Morris-Rosendahl, DJ
    AMERICAN JOURNAL OF HUMAN GENETICS, 2003, 73 (05) : 514 - 514
  • [9] An apamin- and scyllatoxin-insensitive isoform of the human SK3 channel
    Wittekindt, OH
    Visan, V
    Tomita, H
    Imtiaz, F
    Gargus, JJ
    Lehmann-Horn, F
    Grissmer, S
    Morris-Rosendahl, DJ
    MOLECULAR PHARMACOLOGY, 2004, 65 (03) : 788 - 801
  • [10] Physiological role of the dominant-negative KCa3.1 channel isoform in mice thymocytes
    Ohya, Susumu
    Fukuyo, Yuka
    Yamamura, Hisao
    Imaizumi, Yuji
    JOURNAL OF PHARMACOLOGICAL SCIENCES, 2011, 115 : 125P - 125P