H2AX phosphorylation and DNA damage kinase activity are dispensable for herpes simplex virus replication

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作者
Carolyn Botting
Xu Lu
Steven J. Triezenberg
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[1] Van Andel Research Institute,Department of Biology
[2] University of Findlay,undefined
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Herpes simplex virus; DNA damage; H2AX; γH2AX; Ataxia telangiectasia mutated; ATM; ATR; Human foreskin fibroblast;
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