Altered cellular localization and hemichannel activities of KID syndrome associated connexin26 I30N and D50Y mutations

被引：0
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作者：
Hande Aypek
Veysel Bay
Gülistan Meşe
机构：
[1] Department of Molecular Biology and Genetics,
[2] Izmir Institute of Technology,undefined
来源：
BMC Cell Biology | / 17卷
关键词：
Connexin26; Hemichannels; Keratitis-ichthyosis-deafness; Intracellular calcium;
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  • [1] Altered cellular localization and hemichannel activities of KID syndrome associated connexin26 I30N and D50Y mutations
    Aypek, Hande
    Bay, Veysel
    Mese, Gulistan
    [J]. BMC CELL BIOLOGY, 2016, 17
  • [2] Insights on the mechanisms of Ca2+ regulation of connexin26 hemichannels revealed by human pathogenic mutations (D50N/Y)
    Lopez, William
    Gonzalez, Jorge
    Liu, Yu
    Harris, Andrew L.
    Contreras, Jorge E.
    [J]. JOURNAL OF GENERAL PHYSIOLOGY, 2013, 142 (01): : 23 - 35
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