Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice

被引：0

作者：

Philip F Stahel

Michael A Flierl

B Paul Morgan

Ivonne Persigehl

Christiane Stoll

Claudia Conrad

Basel M Touban

Wade R Smith

Kathryn Beauchamp

Oliver I Schmidt

Wolfgang Ertel

Iris Leinhase

机构：

[1] University of Colorado School of Medicine,Department of Orthopedic Surgery, Denver Health Medical Center

[2] School of Medicine,Department of Medical Biochemistry and Immunology

[3] Cardiff University,Department of Trauma and Reconstructive Surgery

[4] Charité University Medical Center,Department of Surgery, Division of Neurosurgery

[5] Campus Benjamin Franklin,undefined

[6] Denver Health Medical Center,undefined

[7] University of Colorado School of Medicine,undefined

[8] Trauma Center St. Georg,undefined

[9] Department of Trauma Surgery and Orthopaedics,undefined

来源：

Journal of Neuroinflammation | / 6卷

关键词：

Traumatic Brain Injury; Neuronal Cell Death; Experimental Allergic Encephalomyelitis; Membrane Attack Complex; Neurological Severity Score;

D O I：

暂无

中图分类号：

学科分类号：

摘要：

引用

共 24 条

[1] Absence of the complement regulatory molecule CD59a leads to exacerbated neuropathology after traumatic brain injury in mice
Stahel, Philip F.
Flierl, Michael A.
Morgan, B. Paul
Persigehl, Ivonne
Stoll, Christiane
Conrad, Claudia
Touban, Basel M.
Smith, Wade R.
Beauchamp, Kathryn
Schmidt, Oliver I.
Ertel, Wolfgang
Leinhase, Iris
JOURNAL OF NEUROINFLAMMATION, 2009, 6
[2] Absence of CD59a leads to exacerbated neuroinflammation and neuropathology after experimental closed head injury in mice
Stahel, Philip F.
Leinhase, Iris
Persigehl, Ivonne
Stoll, Christiane
Ertel, Wolfgang
Morgan, B. Paul
MOLECULAR IMMUNOLOGY, 2007, 44 (16) : 3943 - 3943
[3] Complement regulatory protein CD59a plays a protective role in immune liver injury of trichloroethylene-sensitized BALB/c mice
Wang, Xian
Yu, Yun
Xie, Hai-Bo
Shen, Tong
Zhu, Qi-Xing
ECOTOXICOLOGY AND ENVIRONMENTAL SAFETY, 2019, 172 : 105 - 113
[4] NEUROPATHOLOGY IN APP/PS1 MICE IS EXACERBATED AFTER CHIMERA (CLOSED-HEAD IMPACT MODEL OF ENGINEERED ROTATIONAL ACCELERATION)-INDUCED TRAUMATIC BRAIN INJURY
Cheng, W. H.
Namjoshi, D. R.
Martens, K. M.
Wilkinson, A.
Ward-Able, T.
Hayat, A.
McInnes, K. A.
Cripton, P. A.
Wellington, C. L.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM, 2016, 36 : 292 - 293
[5] EXACERBATED INFLAMMATORY RESPONSES RELATED TO ACTIVATED MICROGLIA AFTER TRAUMATIC BRAIN INJURY IN PROGRANULIN-DEFICIENT MICE
Tanaka, Y.
Matsuwaki, T.
Yamanouchi, K.
Nishihara, M.
NEUROSCIENCE, 2013, 231 : 49 - 60
[6] Differential neuropathology and functional outcome after equivalent traumatic brain injury in aged versus young adult mice
Islam, Mecca B. A. R.
Davis IV, Booker T.
Kando, Mary J.
Mao, Qinwen
Procissi, Daniele
Weiss, Craig
Schwulst, Steven J.
EXPERIMENTAL NEUROLOGY, 2021, 341
[7] INCREASED LYSOSOMAL BIOGENESIS IN ACTIVATED MICROGLIA AND EXACERBATED NEURONAL DAMAGE AFTER TRAUMATIC BRAIN INJURY IN PROGRANULIN-DEFICIENT MICE
Tanaka, Y.
Matsuwaki, T.
Yamanouchi, K.
Nishihara, M.
NEUROSCIENCE, 2013, 250 : 8 - 19
[8] Reduction of inflammatory responses by L-serine treatment leads to neuroprotection in mice after traumatic brain injury
Zhai, Pei-Pei
Xu, Li-Hua
Yang, Juan-Juan
Jiang, Zheng-Lin
Zhao, Guang-Wei
Sun, Li
Wang, Guo-Hua
Li, Xia
NEUROPHARMACOLOGY, 2015, 95 : 1 - 11
[9] Erythropoietin Improves Histological and Functional Outcomes after Traumatic Brain Injury in Mice in the Absence of the Neural Erythropoietin Receptor
Xiong, Ye
Mahmood, Asim
Qu, Changsheng
Kazmi, Humaira
Zhang, Zheng Gang
Noguchi, Constance T.
Schallert, Timothy
Chopp, Michael
JOURNAL OF NEUROTRAUMA, 2010, 27 (01) : 205 - 215
[10] Absence of TLR4 Reduces Neurovascular Unit and Secondary Inflammatory Process after Traumatic Brain Injury in Mice
Ahmad, Akbar
Crupi, Rosalia
Campolo, Michela
Genovese, Tiziana
Esposito, Emanuela
Cuzzocrea, Salvatore
PLOS ONE, 2013, 8 (03):

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