CCL2/CCR2 augments the production of transforming growth factor-beta1, type 1 collagen and CCL2 by human CD45-/collagen 1-positive cells under high glucose concentrations

被引：0
|
作者：
Akinori Hara
Norihiko Sakai
Kengo Furuichi
Yoshio Sakai
Motohiro Takeya
Richard Bucala
Naofumi Mukaida
Yoh Takuwa
Kouji Matsushima
Shuichi Kaneko
Takashi Wada
机构：
[1] Kanazawa University,Department of Disease Control and Homeostasis
[2] Kanazawa University,Division of Nephrology, Department of Laboratory Medicine, Faculty of Medicine, Institute of Medical, Pharmaceutical and Health Sciences
[3] Kanazawa University,Department of Laboratory Medicine, Faculty of Medicine, Institute of Medical, Pharmaceutical and Health Sciences
[4] Kanazawa University,Department of Physiology, Faculty of Medicine, Institute of Medical, Pharmaceutical and Health Sciences
[5] Kanazawa University,Division of Molecular Bioregulation, Cancer Research Institute
[6] Kumamoto University,Department of Cell Pathology, Graduate School of Medical Sciences
[7] Yale University School of Medicine,Department of Medicine
[8] The University of Tokyo,Department of Molecular Preventive Medicine, Graduate School of Medicine
来源：
Clinical and Experimental Nephrology | 2013年 / 17卷
关键词：
Diabetes mellitus; CCR2; Fibrosis; Bone marrow-derived cells; Fibrocyte; CD45;
D O I：
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中图分类号：
学科分类号：
摘要：
引用
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页码：793 / 804
页数：11
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