Epigenetic response in mice mastitis: Role of histone H3 acetylation and microRNA(s) in the regulation of host inflammatory gene expression during Staphylococcus aureus infection

被引：0
|
作者：
Rahul Modak
Susweta Das Mitra
Madavan Vasudevan
Paramanandhan Krishnamoorthy
Manoj Kumar
Akshay V Bhat
Mani Bhuvana
Sankar K Ghosh
Bibek R Shome
Tapas K Kundu
机构：
[1] Molecular Biology and Genetics Unit Jawaharlal Nehru Centre for Advanced Scientific Research,Transcription and Disease Laboratory
[2] Project Directorate on Animal Disease Monitoring and Surveillance,Department of Biotechnology
[3] Bionivid Technology [P] Ltd,School of Biotechnology
[4] Assam University,Department of Biochemistry
[5] Kalinga Institute of Industrial Technology,undefined
[6] Veterinary College and Research Institute,undefined
来源：
Clinical Epigenetics | 2014年 / 6卷
关键词：
Epigenetic modifications; Histone; Acetylation; Mastitis; infection; Micro-RNA; Gene expression; Inflammatory response; Chromatin immunoprecipitation;
D O I：
暂无
中图分类号：
学科分类号：
摘要：
引用
收藏
相关论文
共 20 条
  • [1] Epigenetic response in mice mastitis: Role of histone H3 acetylation and microRNA(s) in the regulation of host inflammatory gene expression during Staphylococcus aureus infection
    Modak, Rahul
    Das Mitra, Susweta
    Vasudevan, Madavan
    Krishnamoorthy, Paramanandhan
    Kumar, Manoj
    Bhat, Akshay V.
    Bhuvana, Mani
    Ghosh, Sankar K.
    Shome, Bibek R.
    Kundu, Tapas K.
    CLINICAL EPIGENETICS, 2014, 6
  • [2] Differential regulation between gene expression and histone H3 acetylation in the variable regions of the TCRP locus
    Wang, LL
    Senoo, M
    Habu, S
    BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2002, 298 (03) : 420 - 426
  • [3] Whole-genome regulation analysis of histone H3 lysin 27 trimethylation in subclinical mastitis cows infected by Staphylococcus aureus
    He, Yanghua
    Song, Minyan
    Zhang, Yi
    Li, Xizhi
    Song, Jiuzhou
    Zhang, Yuan
    Yu, Ying
    BMC GENOMICS, 2016, 17
  • [4] Whole-genome regulation analysis of histone H3 lysin 27 trimethylation in subclinical mastitis cows infected by Staphylococcus aureus
    Yanghua He
    Minyan Song
    Yi Zhang
    Xizhi Li
    Jiuzhou Song
    Yuan Zhang
    Ying Yu
    BMC Genomics, 17
  • [5] MCN5-Mediated histone H3 acetylation plays a key role in gene expression in Plasmodium falciparum
    Cui, Long
    Miao, Jun
    Furuya, Tetsuya
    Li, Xinyi
    Su, Xin-Zhuan
    Cui, Liwang
    EUKARYOTIC CELL, 2007, 6 (07) : 1219 - 1227
  • [6] High Glucose Leads to Increased Inflammatory Gene Expression via Epigenetic Histone H3 Lysine 9 Methylation in Cardiomyocyte
    Yu, Xi-Yong
    Geng, Yong-Jian
    Lin, Qiu-Xiong
    Xia, Hui-Su
    Shan, Zhi-Xin
    Yang, Min
    Lin, Shu-Guang
    Li, Yangxin
    CIRCULATION, 2010, 122 (21)
  • [7] Gene co-expression in response to Staphylococcus aureus infection reveals networks of genes with specific functions during bovine subclinical mastitis
    Wang, Mengqi
    Bissonnette, Nathalie
    Laterriere, Mario
    Dudemaine, Pier-Luc
    Gagne, David
    Roy, Jean-Philippe
    Sirard, Marc -Andre
    Ibeagha-Awemu, Eveline M.
    JOURNAL OF DAIRY SCIENCE, 2023, 106 (08) : 5517 - 5536
  • [8] Epigenetic regulation of histone H3 lysine 27 (H3K27) controls the division-specific timing of gene expression during B cell differentiation
    Scharer, Christopher
    Barwick, Benjamin
    Bally, Alexander
    Boss, Jeremy M.
    JOURNAL OF IMMUNOLOGY, 2016, 196
  • [9] The SaeR/S Gene Regulatory System Induces a Pro-Inflammatory Cytokine Response during Staphylococcus aureus Infection
    Watkins, Robert L.
    Pallister, Kyler B.
    Voyich, Jovanka M.
    PLOS ONE, 2011, 6 (05):
  • [10] Epigenetic Regulation of Condensin-Mediated Genome Organization during the Cell Cycle and upon DNA Damage through Histone H3 Lysine 56 Acetylation
    Tanaka, Atsunari
    Tanizawa, Hideki
    Sriswasdi, Sira
    Iwasaki, Osamu
    Chatterjee, Atreyi G.
    Speicher, David W.
    Levin, Henry L.
    Noguchi, Eishi
    Noma, Ken-ichi
    MOLECULAR CELL, 2012, 48 (04) : 532 - 546
12