Ethanol ingestion increases gelatinase activity and acute edematous injury in lungs isolated from endotoxin-treated rats.

被引：0

作者：

Lois, M

Roman, J

Moss, IM

Guidot, DM

机构：

[1] Emory Univ, Dept Med, Atlanta, GA 30322 USA

[2] Atlanta VAMC, Atlanta, GA USA

来源：

JOURNAL OF INVESTIGATIVE MEDICINE | 1998年 / 46卷 / 01期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

引用

页码：16A / 16A

页数：1

共 15 条

[1] Chronic ethanol ingestion alters surfactant composition and increases acute lung injury in septic rats.
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Guidot, DM
JOURNAL OF INVESTIGATIVE MEDICINE, 2000, 48 (01) : 173A - 173A
[2] Endotoxin tolerance increases resistance to ethanol but not indomethacin induced gastric injury in rats.
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GASTROENTEROLOGY, 1996, 110 (04) : A107 - A107
[3] FIBRONECTIN SYNTHESIS BY HEPATIC KUPFFER CELLS ISOLATED FROM ENDOTOXIN-TREATED RATS
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CIRCULATORY SHOCK, 1987, 21 (04) : 377 - 377
[4] EFFECT OF SERUM FROM ENDOTOXIN-TREATED RATS ON ISOLATED TRYPANOSOMA-EQUIPERDUM
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ACTA BIOLOGICA ACADEMIAE SCIENTIARUM HUNGARICAE, 1971, 22 (01): : 53 - &
[5] COVALENT BINDING OF [THIOUREA-C-14 TO PROTEIN IN LUNGS FROM ENDOTOXIN-TREATED RATS
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[6] Effect of ketamine on NF-kappa B activity and TNF-alpha production in endotoxin-treated rats.
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ANNALS OF CLINICAL AND LABORATORY SCIENCE, 2004, 34 (02): : 181 - 186
[7] Chronic ethanol ingestion impairs alveolar type II cell glutathione homeostasis and function and predisposes to endotoxin-mediated acute edematous lung injury in rats
Holguin, F
Moss, IM
Brown, LAS
Guidot, DM
JOURNAL OF CLINICAL INVESTIGATION, 1998, 101 (04): : 761 - 768
[8] NITRIC-OXIDE CAUSES HYPOREACTIVITY TO PHENYLEPHRINE IN ISOLATED-PERFUSED LIVERS FROM ENDOTOXIN-TREATED RATS
PASTOR, CM
BILLIAR, TR
AMERICAN JOURNAL OF PHYSIOLOGY-GASTROINTESTINAL AND LIVER PHYSIOLOGY, 1995, 268 (01): : G177 - G182
[9] HYPOREACTIVITY TO PHENYLEPHRINE IN ISOLATED-PERFUSED LIVERS FROM ENDOTOXIN-TREATED RATS - REVERSAL BY NITRIC-OXIDE (NO) BLOCKAGE
PASTOR, CM
BILLIAR, TR
ANESTHESIOLOGY, 1994, 81 (3A) : A328 - A328
[10] Effect of modulation of Kupffer cells on tumor necrosis factor-α release into the perfusate of cold stored livers obtained from endotoxin-treated rats.
Lutterova, M
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CHEMICAL PAPERS, 1998, 52 : 465 - 465

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