Stimulus intensity and duration modulate intercellular Ca2+ wave initiation and transmission along endothelium of mouse resistance arteries

被引:0
|
作者
Socha, Matthew [1 ]
Segal, Steven [1 ]
机构
[1] Univ Missouri, Columbia, MO 65211 USA
关键词
D O I
暂无
中图分类号
Q4 [生理学];
学科分类号
071003 ;
摘要
ISRA102
引用
收藏
页码:123 / 123
页数:1
相关论文
共 7 条
  • [1] Coordination of Intercellular Ca2+ Signaling in Endothelial Cell Tubes of Mouse Resistance Arteries
    Socha, Matthew J.
    Domeier, Timothy L.
    Behringer, Erik J.
    Segal, Steven S.
    MICROCIRCULATION, 2012, 19 (08) : 757 - 770
  • [3] Extracellular Histones Induce Propagating Ca2+ Influx, Ca2+ Overload, and Endothelial Cell Death in Resistance-sized Mouse Mesenteric Arteries
    Collier, Daniel M.
    Villalba, Nuria M.
    Sackheim, Adrian M.
    Freeman, Kalev
    Nelson, Mark T.
    FASEB JOURNAL, 2017, 31
  • [4] Smooth muscle gap-junctions allow propagation of intercellular Ca2+ waves and vasoconstriction due to Ca2+ based action potentials in rat mesenteric resistance arteries
    Borysova, Lyudmyla
    Dora, Kim A.
    Garland, Christopher J.
    Burdyga, Theodor
    CELL CALCIUM, 2018, 75 : 21 - 29
  • [5] High intravascular pressure decreases endothelial Ca2+ pulsars and impairs endothelium-dependent vasodilation in mouse mesenteric arteries
    Sonkusare, Swapnil K.
    Heppner, Thomas J.
    Bonev, Adrian D.
    Hannah, Rachael M.
    Talline, Yvonne
    Kotlikoff, Michael I.
    Nelson, Mark T.
    FASEB JOURNAL, 2010, 24
  • [6] Aging Impairs Electrical Conduction Along Endothelium of Resistance Arteries Through Enhanced Ca2+-Activated K+ Channel Activation
    Behringer, Erik J.
    Shaw, Rebecca L.
    Westcott, Erika B.
    Socha, Matthew J.
    Segal, Steven S.
    ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY, 2013, 33 (08) : 1892 - 1901
  • [7] Marfan Syndrome Decreases Ca2+ Wave Frequency and Vasoconstriction in Murine Mesenteric Resistance Arteries without Changing Underlying Mechanisms
    Syyong, H. T.
    Chung, A. W. Y.
    van Breemen, C.
    JOURNAL OF VASCULAR RESEARCH, 2011, 48 (02) : 150 - 162