Neuroimmune interactions in chronic itch of atopic dermatitis

被引：105
|
作者：
Yosipovitch, G. [1 ,2 ]
Berger, T. [3 ]
Fassett, M. S. [3 ]
机构：
[1] Univ Miami, Miller Sch Med, Dept Dermatol, Miami, FL 33136 USA
[2] Univ Miami, Miller Sch Med, Cutaneous Surg & Miami Itch Ctr, Miami, FL 33136 USA
[3] Univ Calif San Francisco, Sch Med, Dept Dermatol, San Francisco, CA USA
来源：
JOURNAL OF THE EUROPEAN ACADEMY OF DERMATOLOGY AND VENEREOLOGY | 2020年 / 34卷 / 02期
关键词：
JAPANESE ADULT PATIENTS; JANUS KINASE INHIBITOR; HUMAN SURROGATE MODELS; NERVE GROWTH-FACTOR; QUALITY-OF-LIFE; DOUBLE-BLIND; SPINAL-CORD; RECEPTOR; PRURITUS; EFFICACY;
D O I：
10.1111/jdv.15973
中图分类号：
R75 [皮肤病学与性病学];
学科分类号：
100206 ;
摘要：
Itch is a defining symptom of atopic dermatitis. Crosstalk between keratinocytes, the immune system and non-histaminergic sensory nerves is responsible for the pathophysiology of chronic itch in atopic dermatitis. An expanding understanding of the contribution of the nervous system and its interaction with immune pathways in atopic itch are helping to identify new therapeutic strategies.
引用
收藏
页码：239 / 250
页数：12
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