Subcutaneous Campath-1H (Alemtuzumab) in fludarabine-refractory CLL interim analysis of the CLL2h study of the German CLL study group (GCLLSG).

被引：0

作者：

Stilgenbauer, S ^{[1
]}

Winkler, D ^{[1
]}

Kröber, A ^{[1
]}

Kienle, D ^{[1
]}

Hallek, M ^{[1
]}

Hensel, M ^{[1
]}

Lengfelder, E ^{[1
]}

Trümper, L ^{[1
]}

Dreger, P ^{[1
]}

Jäger, U ^{[1
]}

Döhner, H ^{[1
]}

机构：

[1] Univ Ulm, Dept Internal Med 3, D-89069 Ulm, Germany

来源：

BLOOD | 2004年 / 104卷 / 11期

关键词：

D O I：

暂无

中图分类号：

R5 [内科学];

学科分类号：

1002 ; 100201 ;

摘要：

478

引用

页码：140A / 140A

页数：1

共 50 条

[1] Efficacy of subcutaneous Alemtuzumab (Campath-1H) in genetic high-risk, fludarabine-refractory CLL:: CLL2H study of the German CLL Study Group (GCLLSG)
Stilgenbauer, S
Kröber, A
Winkler, D
Kienle, D
Busch, R
Hallek, M
Hensel, M
Lengfelder, E
Trümper, L
Dreger, P
Jäger, U
Döhner, H
ANNALS OF ONCOLOGY, 2005, 16 : 43 - 43
[2] Subcutaneous alemtuzumab (MabCampath) in fludarabine-refractory CLL (CLL2H trial of the GCLLSG)
Stilgenbauer, Stephan
Winkler, Dirk
Buehler, Andreas
Zenz, Thorsten
Groner, Sija
Busch, Raymonde
Hensel, Manfred
Duehrsen, Uhich
Finke, Juergen
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Schlag, Rudolf
Hallek, Michael
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[3] Subcutaneous Alemtuzumab (Campath) in Fludarabine-Refractory CLL: Final Results of the CLL2H Trial of the GCLLSG and Comprehensive Analysis of Prognostic Markers
Stilgenbauer, Stephan
Zenz, Thorsten
Winkler, Dirk
Buehler, Andreas
Groner, Sija
Busch, Raymonde
Hensel, Manfred
Duehrsen, Ulrich
Finke, Juergen
Dreger, Peter
Jaeger, Ulrich
Lengfelder, Eva
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[4] The activity of alemtuzumab is independent of p53 mutational status in fludarabine refractory CLL:: Interim analysis from the CLL2H study of the GCLLSG
Zenz, T.
Haebe, S.
Denzel, T.
Winkler, D.
Kroeber, A.
Kienle, D.
Hallek, M.
Hensel, M.
Lengfelder, E.
Truemper, L.
Dreger, P.
Jaeger, U.
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HAEMATOLOGICA-THE HEMATOLOGY JOURNAL, 2007, 92 : 42 - 42
[5] High CD52 mRNA Expression Is An Adverse Prognostic Factor In Fludarabine-Refractory CLL Treated With Alemtuzumab On The Gcllsg CLL2H Trial
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Sill, Martin
Langer, Christian
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[6] The activity of alemtuzumab is independent of P53 mutational status in fludarabine refractory CLL:: Final analysis from the CLL2H study of the GCLLSG
Zenz, T.
Haebe, S.
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Winkler, D.
Hallek, M.
Hensel, M.
Lengfelder, E.
Dreger, P.
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Doehner, H.
Stilgenbauer, S.
ANNALS OF ONCOLOGY, 2008, 19 : 193 - 194
[7] Subcutaneous Alemtuzumab in Fludarabine-Refractory Chronic Lymphocytic Leukemia: Clinical Results and Prognostic Marker Analyses From the CLL2H Study of the German Chronic Lymphocytic Leukemia Study Group
Stilgenbauer, Stephan
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Dreger, Peter
Jaeger, Ulrich
Lengfelder, Eva
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JOURNAL OF CLINICAL ONCOLOGY, 2009, 27 (24) : 3994 - 4001
[8] NOTCH1, SF3B1, and TP53 mutations in fludarabine-refractory CLL patients treated with alemtuzumab: results from the CLL2H trial of the GCLLSG
Schnaiter, Andrea
Paschka, Peter
Rossi, Marianna
Zenz, Thorsten
Buehler, Andreas
Winkler, Dirk
Cazzola, Mario
Doehner, Konstanze
Edelmann, Jennifer
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BLOOD, 2013, 122 (07) : 1266 - 1270
[9] NOTCH1, SF3B1 and TP53 Mutations in Fludarabine-Refractory CLL Patients Treated with Alemtuzumab: Results From the CLL2H Trial of the Gcllsg
Schnaiter, Andrea
Rossi, Marianna
Paschka, Peter
Cazzola, Mario
Doehner, Konstanze
Edelmann, Jennifer
Buehler, Andreas
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Stilgenbauer, Stephan
BLOOD, 2012, 120 (21)
[10] Chemoimmuno-Therapy with Fludarabine, Cyclophosphamide and Alemtuzumab in Patients with Relapsed/Refractory CLL: Interim Analysis of the CLL2L Trial of the German CLL Study Group
Elter, Thomas
James, Rojin
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Ritgen, Matthias
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BLOOD, 2008, 112 (11) : 1087 - 1087

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