An intron splicing enhancer element, UGCAUG, is evolutionarily conserved near erythroid protein 4.1R exon 16 and other tissue-specific alternative exons

被引：0
|
作者：
Gee, SL
Lersch, RL
Minovitsky, S
Ponthier, JL
Lo, AJ
Short, S
Chasis, JA
Dubchak, I
Conboy, JG
机构：
[1] Lawrence Berkeley Natl Lab, Div Life Sci, Berkeley, CA USA
[2] Lawrence Berkeley Natl Lab, Div Genom, Berkeley, CA USA
来源：
BLOOD | 2004年 / 104卷 / 11期
关键词：
D O I：
10.1182/blood.V104.11.1584.1584
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
1584
引用
收藏
页码：441A / 441A
页数：1
相关论文
共 16 条
  • [1] An intron splicing enhancer element, UGCAUG, is evolutionarily conserved near tissue-specific alternative exons including erythroid protein 4.1 exon 16.
    Gee, SL
    Lersch, RA
    Minovitsky, S
    Ponthier, JL
    Lo, AJ
    Short, S
    Chasis, JA
    Dubchak, I
    Conboy, JG
    [J]. BLOOD CELLS MOLECULES AND DISEASES, 2005, 34 (02) : 81 - 81
  • [2] The splicing regulatory element, UGCAUG, is phylogenetically and spatially conserved in introns that flank tissue-specific alternative exons
    Minovitsky, S
    Gee, SL
    Schokrpur, S
    Dubchak, I
    Conboy, JG
    [J]. NUCLEIC ACIDS RESEARCH, 2005, 33 (02) : 714 - 724
  • [3] Fox-2 splicing factor binds to a conserved intron motif to promote inclusion of protein 4.1R alternative exon 16
    Ponthier, JL
    Schluepen, C
    Chen, WG
    Lerscho, RA
    Gee, SL
    Hou, VC
    Lo, AJ
    Short, SA
    Chasis, JA
    Winkelmann, JC
    Conboy, JG
    [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2006, 281 (18) : 12468 - 12474
  • [4] Differential expression of the SR protein splicing factors and alternative splicing of protein 4.1R exon 16 during erythroid maturation.
    Huang, SC
    Baklouti, F
    Hui, C
    Benz, EJ
    [J]. BLOOD, 2000, 96 (11) : 592A - 592A
  • [5] Evolutionarily conserved coupling of transcription and alternative splicing in the protein 4.1R and 4.1B genes regulates N-terminal protein structure.
    Tan, J
    Parra, MK
    Mohandas, N
    Conboy, JG
    [J]. BLOOD, 2005, 106 (11) : 475A - 475A
  • [6] Interaction of SF2/ASF and an exonic splicing enhancer regulates exon 16 splicing in protein 4.1R pre-mRNA.
    Yang, G
    Huang, SC
    Benz, EJ
    [J]. BLOOD, 2003, 102 (11) : 256A - 256A
  • [7] An erythroid differentiation-specific splicing switch in protein 4.1R mediated by the interaction of SF2/ASF with an exonic splicing enhancer
    Yang, G
    Huang, SC
    Wu, JY
    Benz, EJ
    [J]. BLOOD, 2005, 105 (05) : 2146 - 2153
  • [8] Combinatorial regulation of protein 4.1R exon 16 alternative splicing: Modulation of fox-2 activated splicing by other intronic and exonic motifs.
    Gee, Sherry L.
    Schluepen, Christina
    Conboy, John G.
    [J]. BLOOD, 2006, 108 (11) : 163A - 163A
  • [9] Multiple isoforms of HRNBP2, a splicing enhancer for erythroid protein 4.1R exon 16, are encoded by a complex gene via alternative pre-mRNA processing events.
    Conboy, JG
    Ponthier, JL
    Gee, SL
    [J]. BLOOD, 2003, 102 (11) : 7A - 7A
  • [10] Protein 4.1R exon 16 splicing mediated by regulated expression of RBM9 and PTB during erythroid differentiation.
    Yang, Guang
    Huang, Shu-Ching
    Benz, Edward J.
    [J]. BLOOD, 2006, 108 (11) : 162A - 162A
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