Lentiviral Vector-Mediated SHC3 Silencing Exacerbates Oxidative Stress Injury in Nigral Dopamine Neurons by Regulating the PI3K-AKT-FoxO Signaling Pathway in Rats with Parkinson's Disease (Expression of Concern of Vol 49, Pg 971, 2018)

被引：0
|
作者：
Gong, Jian
Zhang, Lei
Zhang, Qian
Li, Xiang
Xia, Xiang-Jun
Liu, Yin-Yuan
Yang, Qin-Shang
机构：
来源：
CELLULAR PHYSIOLOGY AND BIOCHEMISTRY | 2021年 / 55卷 / 05期
关键词：
D O I：
10.33594/000000468
中图分类号：
Q2 [细胞生物学];
学科分类号：
071009 ; 090102 ;
摘要：
引用
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页码：678 / 678
页数：1
相关论文
共 8 条
  • [1] RETRACTION: Lentiviral Vector-Mediated SHC3 Silencing Exacerbates Oxidative Stress Injury in Nigral Dopamine Neurons by Regulating the PI3K-AKT-FoxO Signaling Pathway in Rats with Parkinson's Disease (Retraction of Vol 49, Pg 971, 2018)
    Gong, Jian
    Zhang, Lei
    Zhang, Qian
    Li, Xiang
    Xia, Xiang-Jun
    Liu, Yin-Yuan
    Yang, Qin-Shang
    [J]. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY, 2022, 56 (02) : 235 - 235
  • [2] RETRACTED: Lentiviral Vector-Mediated SHC3 Silencing Exacerbates Oxidative Stress Injury in Nigral Dopamine Neurons by Regulating the PI3K-AKT-FoxO Signaling Pathway in Rats with Parkinson's Disease (Publication with Expression of Concern. See vol. 55, pg. 678, 2021) (Retracted Article)
    Gong, Jian
    Zhang, Lei
    Zhang, Qian
    Li, Xiang
    Xia, Xiang-Jun
    Liu, Yin-Yuan
    Yang, Qin-Shang
    [J]. CELLULAR PHYSIOLOGY AND BIOCHEMISTRY, 2018, 49 (03) : 971 - 984
  • [3] Lentivirus vector mediated SOD1 gene silencing targets the PI3K/AKT signaling pathway to protect dopaminergic neurons from oxidative stress in the substantia nigra of Parkinson's disease rats
    Xie, Chaoyan
    Chen, Qiang
    Xiao, Yuan
    Xie, Zenghua
    Fang, Xing
    [J]. INTERNATIONAL JOURNAL OF CLINICAL AND EXPERIMENTAL MEDICINE, 2019, 12 (09): : 11210 - 11220
  • [4] RETRACTION: Downregulated expression of microRNA-329 inhibits apoptosis of nigral dopaminergic neurons by regulating CDKN2D expression via the FoxO3a signaling pathway in rats with Parkinson's disease (Retraction of Vol 233, Pg 8617, 2018)
    Liu, Yuan-Yuan
    Zhang, Yi-Nan
    Yang, Qing-Shan
    [J]. JOURNAL OF CELLULAR PHYSIOLOGY, 2022, 237 (03) : 1997 - 1997
  • [5] Downregulation of lncRNA UCA1 ameliorates the damage of dopaminergic neurons, reduces oxidative stress and inflammation in Parkinson's disease through the inhibition of the PI3K/Akt signaling pathway (Publication with Expression of Concern. See vol. 114, 2023)
    Cai, Lijun
    Tu, Li
    Li, Tian
    Yang, Xiulin
    Ren, Yipin
    Gu, Ran
    Zhang, Qian
    Yao, Huan
    Qu, Xiang
    Wang, Qian
    Tian, Jinyong
    [J]. INTERNATIONAL IMMUNOPHARMACOLOGY, 2019, 75
  • [6] Downregulation of lncRNA UCA1 ameliorates the damage of dopaminergic neurons, reduces oxidative stress and inflammation in Parkinson's disease through the inhibition of the PI3K/Akt signaling pathway (Expression of Concern of Vol 75, 10.1016/J.INTIMP.2019.105734, 2019)
    Cai, Lijun
    Tu, Li
    Li, Tian
    Yang, Xiulin
    Ren, Yipin
    Gu, Ran
    Zhang, Qian
    Yao, Huan
    Qu, Xiang
    Wang, Qian
    Tian, Jinyong
    [J]. INTERNATIONAL IMMUNOPHARMACOLOGY, 2023, 114
  • [7] Long noncoding RNA HAGLROS regulates apoptosis and autophagy in Parkinson's disease via regulating miR-100/ATG10 axis and PI3K/Akt/mTOR pathway activation (Expression of Concern of Vol 47, Pg 2764, 2019)
    Peng, Tao
    Liu, Xiaoyan
    Wang, Jingtao
    Liu, Yu
    Fu, Zhenqiang
    Ma, Xingrong
    Li, Junmin
    Sun, Guifang
    Ji, Yangfei
    Lu, Jingjing
    Wan, Wencui
    Lu, Hong
    [J]. ARTIFICIAL CELLS NANOMEDICINE AND BIOTECHNOLOGY, 2020, 48 (01) : 708 - 708
  • [8] RETRACTED: Long noncodinq RNA HAGLROS regulates apoptosis and autophagy in Parkinson's disease via regulating miR-100/ATG10 axis and PI3K/Akt/mTOR pathway activation (Publication with Expression of Concern. See vol. 48, pg. 708, 2020) (Retracted article. See vol. 49, pg. 543, 2021)
    Peng, Tao
    Liu, Xiaoyan
    Wang, Jingtao
    Liu, Yu
    Fu, Zhenqiang
    Ma, Xingrong
    Li, Junmin
    Sun, Guifang
    Ji, Yangfei
    Lu, Jingjing
    Wan, Wencui
    Lu, Hong
    [J]. ARTIFICIAL CELLS NANOMEDICINE AND BIOTECHNOLOGY, 2019, 47 (01) : 2764 - 2774
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