ADRENERGIC HYPERFUNCTION IS A KEY TRIGGER OF SOLUTE-FREE WATER RETENTION IN ASCITIC CIRRHOSIS: VASOPRESSIN (ADH) IS NOT THE ONLY AGENT TO BLAME

被引：0

作者：

Sansoe, G. ^{[1
]}

Aragno, M. ^{[2
]}

Mastrocola, R. ^{[2
]}

Rosina, F. ^{[1
]}

Parola, M. ^{[2
]}

机构：

[1] Gradenigo Hosp, Div Gastroenterol, Turin, Italy

[2] Univ Turin, Dept Clin & Biol Sci, Turin, Italy

来源：

DIGESTIVE AND LIVER DISEASE | 2015年 / 47卷

关键词：

D O I：

10.1016/j.dld.2015.01.044

中图分类号：

R57 [消化系及腹部疾病];

学科分类号：

摘要：

T-01

引用

页码：E19 / E19

页数：1

共 7 条

[1] SOLUTE-FREE WATER RETENTION IN EXPERIMENTAL ASCITIC CIRRHOSIS: VASOPRESSIN (ADH) IS NOT THE ONLY AGENT TO BLAME
Sansoe, G.
Aragno, M.
Mastrocola, R.
Parola, M.
JOURNAL OF HEPATOLOGY, 2015, 62 : S381 - S382
[2] Pathogenesis of solute-free water retention in experimental ascitic cirrhosis: is vasopressin (ADH) the only agent to blame?
Sansoe, Giovanni
Aragno, Manuela
Mastrocola, Raffaella
Mengozzi, Giulio
Whomsley, Rhys
Parola, Maurizio
HEPATOLOGY, 2013, 58 : 888A - 888A
[3] PATHOGENESIS OF SOLUTE-FREE WATER RETENTION IN EXPERIMENTAL ASCITIC CIRRHOSIS: IS VASOPRESSIN (ADH) THE ONLY AGENT TO BLAME?
Sansoe, G.
Aragno, M.
Mastrocola, R.
Parola, M.
DIGESTIVE AND LIVER DISEASE, 2014, 46 : E34 - E35
[4] Pathogenesis of solute-free water retention in experimental ascitic cirrhosis: is vasopressin the only culprit?
Sansoe, Giovanni
Aragno, Manuela
Mastrocola, Raffaella
Parola, Maurizio
CLINICAL SCIENCE, 2016, 130 (02) : 117 - 124
[5] PROOF OF MULTIPLE MECHANISMS OF SOLUTE-FREE WATER RETENTION IN EXPERIMENTAL ASCITIC CIRRHOSIS
Sansoe, G.
Aragno, M.
Mastrocola, R.
Parola, M.
JOURNAL OF HEPATOLOGY, 2016, 64 : S658 - S658
[6] Proof of Multiple Mechanisms of Solute-Free Water Retention in Experimental Ascitic Cirrhosis
Sansoe, Giovanni
Mastrocola, Raffaella
Aragno, Manuela
Parola, Maurizio
GASTROENTEROLOGY, 2016, 150 (04) : S1084 - S1084
[7] PROOF OF MULTIPLE MECHANISMS OF SOLUTE-FREE WATER RETENTION IN EXPERIMENTAL ASCITIC CIRRHOSIS
Sansoe, G.
Aragno, M.
Mastrocola, R.
Parola, M.
DIGESTIVE AND LIVER DISEASE, 2016, 48 : E25 - E25

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