Possible functions mediated through the C-terminal repression domain of leukemia-associated transcription factor, acute myeloid leukemia 1 (AML1), in mouse development.

被引:0
|
作者
Nishimura, M
Fujita, Y
Toda, S
Kitamura, N
Abe, T
Okuda, T
机构
[1] Kyoto Prefectural Univ, Dept Hyg, Kyoto 606, Japan
[2] Kyoto Prefectural Univ, Dept Thorac Surg, Kyoto 606, Japan
[3] Kyoto Prefectural Univ, Dept Internal Med, Kyoto 606, Japan
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
1211
引用
收藏
页码:282A / 282A
页数:1
相关论文
共 50 条
  • [1] The acute myeloid leukemia-associated Nucleophosmin 1 gene mutations dictate amyloidogenicity of the C-terminal domain
    La Manna, Sara
    Scognamiglio, Pasqualina Liana
    Roviello, Valentina
    Borbone, Fabio
    Florio, Daniele
    Di Natale, Concetta
    Bigi, Alessandra
    Cecchi, Cristina
    Cascella, Roberta
    Giannini, Cinzia
    Sibillano, Teresa
    Novellino, Ettore
    Marasco, Daniela
    FEBS JOURNAL, 2019, 286 (12) : 2311 - 2328
  • [2] Genetically modified mouse lines with a limited repertoire of the isoforms for leukemia-associated transcription factor, AML1/RUNX1.
    Nishimura, M
    Fujita, Y
    Nakao, M
    Fukushima, Y
    Toda, S
    Kitamura, N
    Abe, T
    Okuda, T
    BLOOD, 2001, 98 (11) : 280A - 281A
  • [3] Differential expression patterns of acute myeloid leukemia 1 (AML1) isoforms in early hematopoietic development.
    Fujita, Y
    Nishimura, M
    Taniwaki, M
    Abe, T
    Okuda, T
    BLOOD, 2000, 96 (11) : 284A - 284A
  • [4] Collaboration of leukemia-associated protein AML1 and PML in myeloid cell differentiation.
    Nguyen, LA
    Aikawa, Y
    Tagata, Y
    Kurihara, K
    Pandolfi, PP
    Ohki, M
    Kitabayashi, I
    BLOOD, 2003, 102 (11) : 220A - 220A
  • [5] Architecture and anatomy of the genomic locus encoding the human leukemia-associated transcription factor RUNX1/AML1
    Levanon, D
    Glusman, C
    Bangsow, T
    Ben-Asher, E
    Male, DA
    Avidan, N
    Bangsow, C
    Hattori, M
    Taylor, TD
    Taudien, S
    Blechschmidt, K
    Shimizu, N
    Rosenthal, A
    Sakaki, Y
    Lancet, D
    Groner, Y
    GENE, 2001, 262 (1-2) : 23 - 33
  • [6] Upstream and downstream of the human leukemia associated transcription factor RUNX1/AML1
    Pozner, A
    Polak, T
    Bangsow, T
    Bernstein, Y
    Negreanu, V
    Goldenberg, D
    Ben Aziz-Aloya, T
    Anghel-Bangsow, C
    Rubins, N
    Bettoun, JD
    Levanon, D
    Groner, Y
    CYTOGENETICS AND CELL GENETICS, 2001, 92 (1-2): : 11 - 12
  • [7] Functional redundancy of the activation subdomains conserved in AML1/RUNX1 leukemia-associated transcription factor family members.
    Fukushima-Nakase, Y
    Nakao, M
    Nishimura, M
    Sugimoto, T
    Okuda, T
    BLOOD, 2002, 100 (11) : 720A - 720A
  • [8] Familial mutations of the transcription factor RUNX1 (AML1, CBFA2) predispose to acute myeloid leukemia
    Ganly, P
    Walker, LC
    Morris, CM
    LEUKEMIA & LYMPHOMA, 2004, 45 (01) : 1 - 10
  • [9] Inhibition of the transforming growth factor β1 signaling pathway by the AML1/ETO leukemia-associated fusion protein
    Jakubowiak, A
    Pouponnot, C
    Berguido, F
    Frank, R
    Mao, SF
    Massagué, J
    Nimer, SD
    JOURNAL OF BIOLOGICAL CHEMISTRY, 2000, 275 (51) : 40282 - 40287
  • [10] The leukemia-associated AML1 (Runx1)–CBFβ complex functions as a DNA-induced molecular clamp
    Jerónimo Bravo
    Zhe Li
    Nancy A. Speck
    Alan J. Warren
    Nature Structural Biology, 2001, 8 : 371 - 378