Analysis of p53 Expression in 84 Cases of Acute Myeloid Leukemia (AML) With Correlation With p53 Mutational Status and AML Subtype

被引:0
|
作者
Fernandez-Pol, Sebastian [1 ]
Ma, Lisa [1 ]
Ohgami, Robert [1 ]
Arber, Daniel [1 ]
机构
[1] Stanford Med Ctr, Dept Pathol, Stanford, CA USA
关键词
D O I
暂无
中图分类号
R-3 [医学研究方法]; R3 [基础医学];
学科分类号
1001 ;
摘要
1376
引用
收藏
页码:344A / 344A
页数:1
相关论文
共 50 条
  • [1] Analysis of p53 Expression in 84 Cases of Acute Myeloid Leukemia (AML) With Correlation With p53 Mutational Status and AML Subtype
    Fernandez-Pol, Sebastian
    Ma, Lisa
    Ohgami, Robert
    Arber, Daniel
    MODERN PATHOLOGY, 2015, 28 : 344A - 344A
  • [2] P53 protein, expression levels are prognostic in AML and predict for mutational status
    Kornblau, Steven M.
    Barnett, Joseph
    Qiu, YiHua
    Chen, Wenjing
    Faderl, Stefan
    Coombes, Kevin R.
    Andreeff, Michael
    BLOOD, 2007, 110 (11) : 708A - 708A
  • [3] Expression of inhibitory (i)ASPP (Apoptosis Stimulating Protein of p53) in acute myeloid leukemia (AML)
    Schittenhelm, M. M.
    Saipi, Bajrami M.
    Kanz, L.
    Kampa-Schittenhelm, K.
    Oncology Research and Treatment, 2015, 38 : 52 - 52
  • [4] The prognostic significance of expression of MDM-2 and its correlation with expression of p53 and p21 in patients with acute myeloid leukemia (AML).
    Faderi, S
    Kantarjian, HM
    Estey, E
    Cortes, J
    Manshouri, T
    Estrov, Z
    Thomas, D
    Pierce, S
    Albitar, M
    BLOOD, 1999, 94 (10) : 74A - 74A
  • [5] p53 Immunohistochemistry (IHC) Predicts High Risk Cytogenetics in Acute Myeloid Leukemia (AML)
    Liebman, Mira
    Craddock, Kenneth
    Schuh, Andre
    Minden, Mark
    Porwit, Anna
    MODERN PATHOLOGY, 2015, 28 : 359A - 359A
  • [6] p53 Immunohistochemistry (IHC) Predicts High Risk Cytogenetics in Acute Myeloid Leukemia (AML)
    Liebman, Mira
    Craddock, Kenneth
    Schuh, Andre
    Minden, Mark
    Porwit, Anna
    LABORATORY INVESTIGATION, 2015, 95 : 359A - 359A
  • [7] P21 and P53 proteins expression in patients with acute myeloid leukemia (AML) and prediction of response to treatment.
    Sthel, VM
    Yamamoto, M
    Kimura, EYS
    Bordin, JO
    Bahia, DMM
    Kerbauy, J
    BLOOD, 2002, 100 (11) : 257B - 257B
  • [8] p53 pathway dysfunction is highly prevalent in acute myeloid leukemia independent of TP53 mutational status
    Quintas-Cardama, A.
    Hu, C.
    Qutub, A.
    Qiu, Y. H.
    Zhang, X.
    Post, S. M.
    Zhang, N.
    Coombes, K.
    Kornblau, S. M.
    LEUKEMIA, 2017, 31 (06) : 1296 - 1305
  • [9] p53 pathway dysfunction is highly prevalent in acute myeloid leukemia independent of TP53 mutational status
    A Quintás-Cardama
    C Hu
    A Qutub
    Y H Qiu
    X Zhang
    S M Post
    N Zhang
    K Coombes
    S M Kornblau
    Leukemia, 2017, 31 : 1296 - 1305
  • [10] Mutational analysis of p53 in T-acute lymphoblastic leukemia
    Nomdedéu, JF
    Badell, I
    Estivill, C
    del Río, E
    Sierra, J
    Baiget, M
    MEDICINA CLINICA, 2000, 115 (15): : 573 - 575