Tunicamycin-induced ER stress in breast cancer cells neither expresses GRP78 on the surface nor secretes it into the media (vol 28, pg 61, 2018)

被引：3
|
作者：
Serrano-Negron, Jesus E.
Zhang, Zhenbo
Rivera-Ruiz, Andrea P.
Banerjee, Aditi
Romero-Nutz, Eva C.
Sanchez-Torres, Neysharie
Baksi, Krishna
Banerjee, Dipak K.
机构：
来源：
GLYCOBIOLOGY | 2019年 / 29卷 / 07期
关键词：
D O I：
10.1093/glycob/cwz030
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
引用
收藏
页码：599 / 599
页数：1
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  • [1] Tunicamycin-induced ER stress in breast cancer cells neither expresses GRP78 on the surface nor secretes it into the media
    Serrano-Negron, Jesus E.
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    Rivera-Ruiz, Andrea P.
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    GLYCOBIOLOGY, 2018, 28 (02) : 61 - 68
  • [2] GRP78 is neither expressed on ER-/PR-/Her2- human breast cancer cell surface nor secreted in the culture media
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    CANCER RESEARCH, 2017, 77
  • [3] Nicotine suppresses tunicamycin-induced, but not thapsigargin-induced, expression of GRP78 during ER stress-mediated apoptosis in PC12 cells
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  • [5] Upregulated Expression of GRP78 Supports ER Stress-induced Apoptosis in Tunicamycin Treated Triple Negative Breast Tumor
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  • [6] Stress protein GRP78 prevents apoptosis induced by calcium ionophore, lonomycin, but not by glycosylation inhibitor, tunicamycin, in human prostate cancer cells
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  • [9] Inhibition of MEK blocks GRP78 up-regulation and enhances apoptosis induced by ER stress in gastric cancer cells
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