Cerebral Aβ deposition is dependent on the-491 AA promoter polymorphism in nondemented elderly bearing the APOE ε4 allele

被引：0
|
作者：
Pahnke, J
Walker, L
Schroeder, E
Vogelgesang, S
Warzok, R
Walther, R
机构：
[1] Univ Rostock, Neurobiol Lab, Neurol Clin, D-2500 Rostock 1, Germany
[2] CNS Pharmacol, Ann Arbor, MI USA
[3] Univ Greifswald, Inst Pathol, D-17487 Greifswald, Germany
[4] Univ Greifswald, Inst Biochem, D-17487 Greifswald, Germany
来源：
NEUROBIOLOGY OF AGING | 2002年 / 23卷 / 01期
关键词：
D O I：
暂无
中图分类号：
R592 [老年病学]; C [社会科学总论];
学科分类号：
03 ; 0303 ; 100203 ;
摘要：
1433
引用
收藏
页码：S391 / S391
页数：1
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  • [1] Cerebral β-amyloid deposition is augmented by the-491AA promoter polymorphism in non-demented elderly individuals bearing the apolipoprotein E ε4 allele
    Pahnke, J
    Walker, LC
    Schroeder, E
    Vogelgesang, S
    Stausske, D
    Walther, R
    Warzok, RW
    [J]. ACTA NEUROPATHOLOGICA, 2003, 105 (01) : 25 - 29
  • [2] Cerebral β-amyloid deposition is augmented by the –491AA promoter polymorphism in non-demented elderly individuals bearing the apolipoprotein E ε4 allele
    J. Pahnke
    L. Walker
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    S. Vogelgesang
    D. Stausske
    R. Walther
    R. Warzok
    [J]. Acta Neuropathologica, 2003, 105 : 25 - 29
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    [J]. HUMAN BRAIN MAPPING, 2015, 36 (12) : 4847 - 4858
  • [5] ApoE −491A/T Promoter Polymorphism is not an Independent Risk Factor, but Associated with the ε4 Allele in Hungarian Alzheimer’s Dementia Population
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    Ágnes Rimanóczy
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    [J]. JOURNAL OF NEUROTRAUMA, 2007, 24 (12) : 1802 - 1810
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    [J]. NEUROLOGY, 2007, 68 (12) : A328 - A328
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