Increased sensitivity to cell death in immortal DF-1 cells by oxidative stress is mediated by overproduction of H2O2 through active mitochondrial and MnSOD functions, not by the p53 tumor suppressor

被引：0

作者：

Kim, H ^{[1
]}

Foster, DN ^{[1
]}

机构：

[1] Univ Minnesota, Minneapolis, MN 55455 USA

来源：

MOLECULAR BIOLOGY OF THE CELL | 1999年 / 10卷

关键词：

D O I：

暂无

中图分类号：

Q2 [细胞生物学];

学科分类号：

071009 ; 090102 ;

摘要：

1912

引用

页码：331A / 331A

页数：1

共 14 条

[1] Increased H2O2 levels and p53 stabilization lead to mitochondrial dysfunction in XPC-deficient cells
Freire, T. S.
Mori, M. P.
Miranda, J. N. F. A.
Muta, L. Y. M.
Machado, F. T.
Moreno, N. C.
Souza-Pinto, N. C.
CARCINOGENESIS, 2021, 42 (11) : 1380 - 1389
[2] Expression of HSP27 results in increased sensitivity to tumor necrosis factor, etoposide, and H2O2 in an oxidative stress-resistant cell line
Mairesse, N
Del Bino, G
Horman, S
Mosselmans, R
Robaye, B
Galand, P
JOURNAL OF CELLULAR PHYSIOLOGY, 1998, 177 (04) : 606 - 617
[3] DEAD/H Box 5 (DDX5) Augments E2F1-Induced Cell Death Independent of the Tumor Suppressor p53
Nakajima, Rinka
Zhou, Yaxuan
Shirasawa, Mashiro
Nishimura, Naoyasu
Zhao, Lin
Fikriyanti, Mariana
Kamiya, Yuki
Iwanaga, Ritsuko
Bradford, Andrew P.
Shinmyozu, Kaori
Nishibuchi, Gohei
Nakayama, Jun-ichi
Kurayoshi, Kenta
Araki, Keigo
Ohtani, Kiyoshi
INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES, 2024, 25 (24)
[4] ABCG1 Attenuates Oxidative Stress Induced by H2O2 through the Inhibition of NADPH Oxidase and the Upregulation of Nrf2-Mediated Antioxidant Defense in Endothelial Cells
Xue, Jiahong
Fan, Jiali
Li, Yuan
Wu, Wenhuan
Yan, Qing
Zheng, Qiangsun
ANALYTICAL CELLULAR PATHOLOGY, 2020, 2020
[5] Mutations in amyloid precursor protein and presenilin-1 genes increase the basal oxidative stress in murine neuronal cells and lead to increased sensitivity to oxidative stress mediated by amyloid β-peptide (1-42), H2O2 and kainic acid:: implications for Alzheimer's disease
Abdul, HM
Sultana, R
Keller, JN
St Clair, DK
Markesbery, WR
Butterfield, DA
JOURNAL OF NEUROCHEMISTRY, 2006, 96 (05) : 1322 - 1335
[6] Inhibition of H2O2-induced TM3 cell apoptosis by oxidative stress by lentinan functionalized selenium nanoparticles through JAK2/STAT-3 and P53 pathways
LI, M. I. A. O. M. I. A. O.
Zheng, Z. I. L. I. N.
Ke, J. U. N. Y., I
Luo, J. I. E. Y. I.
Jiang, F. A. N.
Qu, Y. A. N. X. I. A.
Zhu, B. I. N. G.
LI, Y. I. N. G. H. U. A.
Zuo, L. I. A. N. D. O. N. G.
BIOCELL, 2023, 47 (06) : 1397 - 1405
[7] Synthesis, characterization and antitumor activity of novel tetrapodal 1,4-dihydropyridines: p53 induction, cell cycle arrest and low damage effect on normal cells induced by genotoxic factor H2O2
Mohamed, Magda F.
Darweesh, Ahmed F.
Elwahy, Ahmed H. M.
Abdelhamid, Ismail A.
RSC ADVANCES, 2016, 6 (47): : 40900 - 40910
[8] Silencing of RUNX2 enhances gemcitabine sensitivity of p53-deficient human pancreatic cancer AsPC-1 cells through the stimulation of TAp63-mediated cell death
Sugimoto, H.
Nakamura, M.
Yoda, H.
Hiraoka, K.
Shinohara, K.
Sang, M.
Fujiwara, K.
Shimozato, O.
Nagase, H.
Ozaki, T.
CELL DEATH & DISEASE, 2015, 6 : e1914 - e1914
[9] Silencing of RUNX2 enhances gemcitabine sensitivity of p53-deficient human pancreatic cancer AsPC-1 cells through the stimulation of TAp63-mediated cell death
Sugimoto, H.
Nakamura, M.
Yoda, H.
Hiraoka, K.
Shinohara, K.
Sang, M.
Fujiwara, K.
Shimozato, O.
Nagase, H.
Ozaki, T.
CELL DEATH DISCOVERY, 2015, 1
[10] Silencing of RUNX2 enhances gemcitabine sensitivity of p53-deficient human pancreatic cancer AsPC-1 cells through the stimulation of TAp63-mediated cell death
H Sugimoto
M Nakamura
H Yoda
K Hiraoka
K Shinohara
M Sang
K Fujiwara
O Shimozato
H Nagase
T Ozaki
Cell Death & Disease, 2015, 6 : e1914 - e1914

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