Genistein selectively depolymerizes apical membrane associated actin during activation of CFTR chloride channels.

被引:0
|
作者
Lehrich, R
Drab, M
Aller, S
Forrest, JN
机构
[1] YALE UNIV,SCH MED,DEPT MED,DIV NEPHROL,NEW HAVEN,CT 06510
[2] MAX DELBRUCK CTR MOL MED,BERLIN,GERMANY
[3] MDIBL,SALSBURY COVE,ME
关键词
D O I
暂无
中图分类号
R5 [内科学]; R69 [泌尿科学(泌尿生殖系疾病)];
学科分类号
1002 ; 100201 ;
摘要
引用
收藏
页码:A2048 / A2048
页数:1
相关论文
共 4 条
  • [1] Chloride secretagogues selectively regulate CFTR trafficking and the apical actin network.
    Lehrich, R
    Aller, SG
    Marino, CR
    Forrest, JN
    JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY, 1996, 7 (09): : A0186 - A0186
  • [2] CYCLIC AMP-STIMULATED APICAL MEMBRANE AMPLIFICATION IN TURTLE BLADDER BETA-CELLS - ACTIVATION OF CHLORIDE CHANNELS
    STETSON, DL
    KIDNEY INTERNATIONAL, 1988, 33 (01) : 427 - 427
  • [3] The cystic fibrosis mutation G1349D within the signature motif LSHGH of NBD2 abolishes the activation of CFTR chloride channels by genistein
    Melin, P
    Thoreau, V
    Norez, C
    Bilan, F
    Kitzis, A
    Becq, F
    BIOCHEMICAL PHARMACOLOGY, 2004, 67 (12) : 2187 - 2196
  • [4] SPECTRIN IS ASSOCIATED WITH MEMBRANE-BOUND ACTIN-FILAMENTS IN PLATELETS AND IS HYDROLYZED BY THE CA-2+-DEPENDENT PROTEASE DURING PLATELET ACTIVATION
    FOX, JEB
    REYNOLDS, CC
    MORROW, JS
    PHILLIPS, DR
    BLOOD, 1987, 69 (02) : 537 - 545