Use of arsentic trioxide (As2O3) in the treatment of acute promyelocytic leukemia (APL) .1. Arsenic causes both apoptosis and partial differentiation of NB4 and fresh APL cells in vitro and in vivo

被引:0
|
作者
Chen, Z
Chen, GO
Shi, XG
Tang, W
Zhu, J
Xiong, SM
Ni, IH
Gazin, C
Waxman, S
Wang, ZY
Chen, SJ
机构
[1] SHANGHAI MED UNIV 2,RUI JIN HOSP,SHANGHAI INST HEMATOL,SHANGHAI,PEOPLES R CHINA
[2] HOP ST LOUIS,PARIS,FRANCE
[3] MT SINAI MED CTR,NEW YORK,NY 10029
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
引用
下载
收藏
页码:864 / 864
页数:1
相关论文
共 42 条
  • [1] Use of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia (APL) .1. As2O3 exerts dose-dependent dual effects on APL cells
    Chen, GQ
    Shi, XG
    Tang, W
    Xiong, SM
    Zhu, J
    Cai, X
    Han, ZG
    Ni, JH
    Shi, GY
    Jia, PM
    Liu, MM
    He, KL
    Niu, C
    Ma, J
    Zhang, P
    Zhang, TD
    Paul, P
    Naoe, T
    Kitamura, K
    Miller, W
    Waxman, S
    Wang, ZY
    deThe, H
    Chen, SJ
    Chen, Z
    BLOOD, 1997, 89 (09) : 3345 - 3353
  • [2] Use of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia (APL).: The MD Anderson experience.
    Lazo, G
    Cortes, J
    O'Brien, S
    Estey, E
    Thomas, D
    Kantarjian, H
    BLOOD, 2001, 98 (11) : 595A - 596A
  • [3] Chloroquine aggravates the arsenic trioxide (As2O3)-induced apoptosis of acute promyelocytic leukemia NB4 cells via inhibiting lysosomal degradation in vitro
    Liu, D. -M
    Zhang, X. -D
    Yang, L.
    EUROPEAN REVIEW FOR MEDICAL AND PHARMACOLOGICAL SCIENCES, 2018, 22 (19) : 6412 - 6421
  • [4] The in vitro study of apoptosis induced with arsenic trioxide (As2O3) and other Chinese medications in acute promyelocytic leukemia (APL) and chronic myelocytic leukemia (CML).
    Liu, JW
    Jun, M
    Ying, W
    BLOOD, 1998, 92 (10) : 193B - 193B
  • [5] The use of arsenic trioxide (As2O3) in the treatment of acute promyelocytic leukemia
    Zhang, P
    JOURNAL OF BIOLOGICAL REGULATORS AND HOMEOSTATIC AGENTS, 1999, 13 (04): : 195 - 200
  • [6] Pathologic, cytogenetic and molecular assessment of acute promyelocytic leukemia (APL) patients treated with arsenic trioxide (As2O3,)
    Zhang, T
    Westervelt, P
    Hess, JL
    LABORATORY INVESTIGATION, 1999, 79 (01) : 150A - 150A
  • [7] Clinical observations on arsenic trioxide (As2O3) and all-trans retinoic acid (ATRA) in the treatment of acute promyelocytic leukemia (APL).
    Ma, J
    Liu, JW
    Chen, LY
    Liang, H
    Zhang, Y
    BLOOD, 1998, 92 (10) : 483A - 483A
  • [8] Arsenic trioxide (As2O3) induced selective apoptosis in acute promyelocytic leukemia (APL) cells involves a hydrogen peroxide (H2O2) dependent pathway.
    Jing, Y
    Dai, J
    Cohen, M
    Weinberg, RS
    Chalmers-Redmen, RME
    Tatton, WG
    Waxman, S
    BLOOD, 1998, 92 (10) : 595A - 595A
  • [9] Apoptosis and differentiation induced by all-trans retinoic acid in acute promyelocytic leukemia (APL) cells in vivo and in APL cell line, UF-1, in vitro
    Sato, A
    Imaizumi, M
    Kizaki, M
    Chikaoka, C
    Saito, T
    Yoshinari, M
    Suzuki, H
    Iinuma, K
    EXPERIMENTAL HEMATOLOGY, 1999, 27 (07) : 48 - 48
  • [10] Assessment of arsenic trioxide (AS2O3) effect on telomerase activity in acute promyelocytic leukaemia patients and NB4 cell line
    Ghaffari, SH
    Jamialahmadi, AH
    Shayan-Asl, N
    Alimoghaddam, K
    Ghavamzadeh, A
    BONE MARROW TRANSPLANTATION, 2006, 37 : S123 - S123