Time isn't kind to female T cells

被引：2
|
作者：
McGill, Cassandra J. [1 ,2 ]
Benayoun, Berenice A. [1 ,3 ,4 ,5 ,6 ]
机构：
[1] Univ Southern Calif, Leonard Davis Sch Gerontol, Los Angeles, CA 90007 USA
[2] Univ Southern Calif, Grad Program Biol Aging, Los Angeles, CA 90007 USA
[3] USC Dornsife Coll Letters Arts & Sci, Mol & Computat Biol Dept, Los Angeles, CA 90007 USA
[4] USC Keck Sch Med, Biochem & Mol Med Dept, Los Angeles, CA 90007 USA
[5] USC Norris Comprehens Canc Ctr, Epigenet & Gene Regulat, Los Angeles, CA 90007 USA
[6] USC Stem Cell Initiat, Los Angeles, CA 90007 USA
来源：
NATURE AGING | 2022年 / 2卷 / 03期
关键词：
D O I：
10.1038/s43587-022-00185-0
中图分类号：
Q2 [细胞生物学];
学科分类号：
071009 ; 090102 ;
摘要：
While investigating sex differences in T cell aging, Mkhikian et al. identified a role for excessive IL-7 signaling and N-glycan branching in age-related T cell dysfunction in women and female mice. These findings point to the increasingly recognized importance of the effects of biological sex on immune aging, and delineate new targetable pathways in age-related immune dysfunction.
引用
收藏
页码：189 / 191
页数：3
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