Exercise-Induced Bronchoconstriction and the Air We Breathe

被引:25
|
作者
Rundell, Kenneth W. [1 ]
Smoliga, James M. [2 ]
Bougault, Valerie [3 ]
机构
[1] Geisinger Commonwealth Sch Med, Dept Basic Sci, 525 Pine St, Scranton, PA 18510 USA
[2] High Point Univ, Dept Phys Therapy, 1 Univ Pkwy, High Point, NC 27262 USA
[3] Univ Lille, Hlth Soc URePSSS, Pluridisciplinary Res Unit Sport, EA7369, Lille, France
关键词
Air pollution; Particulate matter; Ozone; Trichloramines; Nitrogen dioxide; Sulfur dioxide; Exercise-induced bronchoconstriction; Asthma; PPM NITROGEN-DIOXIDE; SWIMMING POOL ATTENDANCE; SHORT-TERM EXPOSURE; ORAL N-ACETYLCYSTEINE; CROSS-COUNTRY SKIERS; ULTRAFINE PARTICLE DEPOSITION; DISINFECTION BY-PRODUCTS; FINE PARTICULATE MATTER; PULMONARY-FUNCTION; SULFUR-DIOXIDE;
D O I
10.1016/j.iac.2018.01.009
中图分类号
R392 [医学免疫学];
学科分类号
100102 ;
摘要
An association between airway dysfunction and airborne pollutant inhalation exists. Volatilized airborne fluorocarbons in ski wax rooms, particulate matter, and trichloromines in indoor environments are suspect to high prevalence of exercise-induced bronchoconstriction and new-onset asthma in athletes competing in cross-country skiing, ice rink sports, and swimming. Ozone is implicated in acute decreases in lung function and the development of new-onset asthma from exposure during exercise. Mechanisms and genetic links are proposed for pollution-related new-onset asthma. Oxidative stress from airborne pollutant inhalation is a common thread to progression of airway damage. Key pollutants and mechanisms for each are discussed.
引用
收藏
页码:183 / 204
页数:22
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