Activated K-ras and INK4a/Arf Deficiency Cooperate During the Development of Pancreatic Cancer by Activation of Notch and NF-κB Signaling Pathways (vol 6, e20537, 2011)

被引：0

作者：

Wang, Z.

机构：

来源：

PLOS ONE | 2014年 / 9卷 / 06期

关键词：

D O I：

10.1371/journal.pone.0101032

中图分类号：

O [数理科学和化学]; P [天文学、地球科学]; Q [生物科学]; N [自然科学总论];

学科分类号：

07 ; 0710 ; 09 ;

摘要：

引用

页数：2

共 11 条

[1] RETRACTION: Activated K-ras and INK4a/Arf Deficiency Cooperate During the Development of Pancreatic Cancer by Activation of Notch and NF-κB Signaling Pathways (Retraction of Vol 6, art no E20537, 2011)
不详
PLOS ONE, 2018, 13 (10):
[2] RETRACTED: Activated K-ras and INK4a/Arf Deficiency Cooperate During the Development of Pancreatic Cancer by Activation of Notch and NF-κB Signaling Pathways (Retracted Article)
Wang, Zhiwei
Banerjee, Sanjeev
Ahmad, Aamir
Li, Yiwei
Azmi, Asfar S.
Gunn, Jason R.
Kong, Dejuan
Bao, Bin
Ali, Shadan
Gao, Jiankun
Mohammad, Ramzi M.
Miele, Lucio
Korc, Murray
Sarkar, Fazlul H.
PLOS ONE, 2011, 6 (06):
[3] Activated Kras and INK4a/Arf deficiency cooperate to produce pancreatic cancer involved with Notch and NF-κB pathways
Wang, Zhiwei
Banerjee, Sanjeev
Ahmad, Aamir
Azmi, Asfar S.
Bao, Bin
Ali, Shadan
Mohammad, Ramzi M.
Miele, Lucio
Korc, Murray
Sarkar, Faziul H.
CANCER RESEARCH, 2011, 71
[4] Constitutive Ras signaling and Ink4a/Arf inactivation cooperate during the development of B-ALL in mice
Sewastianik, Tomasz
Jiang, Meng
Sukhdeo, Kumar
Patel, Sanjay S.
Roberts, Kathryn
Kang, Yue
Alduaij, Ahmad
Dennis, Peter S.
Lawney, Brian
Liu, Ruiyang
Song, Zeyuan
Xiong, Jessie
Zhang, Yunyu
Lemieux, Madeleine E.
Pinkus, Geraldine S.
Rich, Jeremy N.
Weinstock, David M.
Mullighan, Charles G.
Sharpless, Norman E.
Carrasco, Ruben D.
BLOOD ADVANCES, 2017, 1 (25) : 2361 - 2374
[5] Activated K-Ras and INK4a/Arf Deficiency Promote Aggressiveness of Pancreatic Cancer by Induction of EMT Consistent With Cancer Stem Cell Phenotype
Wang, Z.
Ali, S.
Banerjee, S.
Bao, B.
Li, Y.
Azmi, A. S.
Korc, M.
Sarkar, F. H.
JOURNAL OF CELLULAR PHYSIOLOGY, 2014, 229 (08) : 1118 - 1118
[6] Constitutive Ras signaling and Ink4a/Arf inactivation cooperate during the development of B-ALL in mice (vol 1, pg 2361, 2017)
Sewastianik, T.
Jiang, M.
Sukhdeo, K.
BLOOD ADVANCES, 2024, 8 (22) : 5878 - 5879
[7] RETRACTION: Activated K-Ras and INK4a/Arf Deficiency Promote Aggressiveness of Pancreatic Cancer by Induction of EMT Consistent With Cancer Stem Cell Phenotype (Retraction of vol 228, pg 556, 2013)
Wang, Z.
Ali, S.
Banerjee, S.
Bao, B.
Li, Y.
Azmi, A. S.
Korc, M.
Sarkar, F. H.
JOURNAL OF CELLULAR PHYSIOLOGY, 2016, 231 (10) : 2304 - 2304
[8] RETRACTED: Activated K-Ras and INK4a/Arf deficiency promote aggressiveness of pancreatic cancer by induction of EMT consistent with cancer stem cell phenotype (Retracted article. See vol. 231, pg. 2304, 2016)
Wang, Zhiwei
Ali, Shadan
Banerjee, Sanjeev
Bao, Bin
Li, Yiwei
Azmi, Asfar S.
Korc, Murray
Sarkar, Fazlul H.
JOURNAL OF CELLULAR PHYSIOLOGY, 2013, 228 (03) : 556 - 562
[9] RETRACTION: Inactivation of Ink4a/Arf Leads to Deregulated Expression of miRNAs in K-Ras Transgenic Mouse Model of Pancreatic Cancer (Retraction of vol 227, pg 3373, 2012)
Ali, S.
Banerjee, S.
Logna, F.
Bao, B.
Philip, P. A.
Korc, M.
Sarkar, F. H.
JOURNAL OF CELLULAR PHYSIOLOGY, 2016, 231 (10) : 2303 - 2303
[10] The mucin MUC4 is a transcriptional and post-transcriptional target of K-ras oncogene in pancreatic cancer. Implication of MAPK/AP-1, NF-κB and RalB signaling pathways
Vasseur, Romain
Skrypek, Nicolas
Duchene, Belinda
Renaud, Florence
Martinez-Maqueda, Daniel
Vincent, Audrey
Porchet, Nicole
Van Seuningen, Isabelle
Jonckheere, Nicolas
BIOCHIMICA ET BIOPHYSICA ACTA-GENE REGULATORY MECHANISMS, 2015, 1849 (12): : 1375 - 1384

← 1 2 →