Wnt/β-Catenin Pathway Contributions to Dendritic Spine and Glutamatergic Synapse Formation Responsive to Lithium-Mediated GSK3 Inhibition

被引:0
|
作者
Stanley, Robert [1 ]
Martin, Pierre-Marie [1 ]
Ross, Adam [1 ]
Freitas, Andiara [1 ]
Iafrati, Jillian [1 ]
Moyer, Caitlin [2 ]
Brumback, Audrey [1 ]
Pirooznia, Mehdi [3 ]
McCombie, W. Richard [4 ]
Potash, James [5 ]
Zandi, Peter [3 ]
Purcell, Shaun [6 ]
Sanders, Stephan [1 ]
Zuo, Yi [2 ]
Sohal, Vikaas [1 ]
Cheyette, Benjamin [1 ]
机构
[1] UCSF, San Francisco, CA USA
[2] UCSC, Santa Cruz, CA USA
[3] Johns Hopkins Univ, Baltimore, MD 21218 USA
[4] Cold Spring Harbor Lab, Stanley Inst, POB 100, Cold Spring Harbor, NY 11724 USA
[5] Univ Iowa, Iowa City, IA 52242 USA
[6] Harvard, Cambridge, MA USA
关键词
WNT signaling; Lithium associated molecular changes; GSK-3; beta; Affective Disorders; Mouse model;
D O I
10.1016/j.biopsych.2017.02.773
中图分类号
Q189 [神经科学];
学科分类号
071006 ;
摘要
706
引用
收藏
页码:S286 / S286
页数:1
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