p16(INK4a) gene inactivation by deletions, mutations, or hypermethylation is associated with aggressive and transformed lymphoproliferative disorders.

被引：0
|
作者：
Cobo, F
Pinyol, M
Hernandez, L
Jares, P
Hernandez, S
Bea, S
Bosch, F
LopezGuillermo, A
Nayach, I
Peiro, N
Costa, D
Fernandez, PL
Campo, E
Montserrat, E
机构：
[1] UNIV BARCELONA,HOSP CLIN,SCH HEMATOL FARRERAS VALENTI,HEMATOPATHOL UNIT,DEPT PATHOL,E-08007 BARCELONA,SPAIN
[2] UNIV BARCELONA,HOSP CLIN,SCH HEMATOL FARRERAS VALENTI,HEMATOPATHOL UNIT,DEPT HEMATOL,E-08007 BARCELONA,SPAIN
来源：
BLOOD | 1997年 / 90卷 / 10期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：2188 / 2188
页数：1
相关论文
共 50 条
  • [1] p16INK4a gene inactivation by deletions, mutations, and hypermethylation is associated with transformed and aggressive variants of non-Hodgkin's lymphomas
    Pinyol, M
    Cobo, F
    Bea, S
    Jares, P
    Nayach, I
    Fernandez, PL
    Montserrat, E
    Cardesa, A
    Campo, E
    BLOOD, 1998, 91 (08) : 2977 - 2984
  • [2] Expression of p16/INK4a in posttransplantation lymphoproliferative disorders
    Martin, A
    Baran-Marzak, F
    El Mansouri, S
    Legendre, C
    Leblond, V
    Charlotte, F
    Davi, F
    Canioni, D
    Raphaël, M
    AMERICAN JOURNAL OF PATHOLOGY, 2000, 156 (05): : 1573 - 1579
  • [3] P16(INK4A) AND P15(INK4B) GENE DELETIONS IN PRIMARY LEUKEMIAS
    HAIDAR, MA
    CAO, XB
    MANSHOURI, T
    CHAN, LL
    GLASSMAN, A
    KANTARJIAN, HM
    KEATING, MJ
    BERAN, MS
    ALBITAR, M
    BLOOD, 1995, 86 (01) : 311 - 315
  • [4] Alterations in the tumor suppressor gene p16 INK4A are associated with aggressive behavior of penile carcinomas
    Poetsch, Micaela
    Hemmerich, Martin
    Kakies, Christoph
    Kleist, Britta
    Wolf, Eduard
    vom Dorp, Frank
    Hakenberg, Oliver W.
    Protzel, Chris
    VIRCHOWS ARCHIV, 2011, 458 (02) : 221 - 229
  • [5] p16(ink4a) gene and hematological malignancies
    Quesnel, B
    Preudhomme, C
    Fenaux, P
    LEUKEMIA & LYMPHOMA, 1996, 22 (1-2) : 11 - 24
  • [6] Epigenetic Silencing of P16 (INK4a) Gene by Promoter Hypermethylation in Chronic Myelogenous Leukemia
    Ahmad, Imtiyaz
    Mir, Rashid
    Javid, Jamsheed
    Farooq, Shazia
    Yadav, Prasant
    Zuberi, Mariyam
    Masroor, M.
    Gupta, Naresh
    Ray, P. C.
    Saxena, Alpana
    CLINICAL LYMPHOMA MYELOMA & LEUKEMIA, 2014, 14 : S139 - S139
  • [7] Promoter hypermethylation of the p16 (CDNK2a/INK4a) gene in lung cancer
    Hughes, S
    Su, XB
    Safar, MA
    Schaefer, RF
    Scott, M
    Smoller, BR
    Fan, CY
    LABORATORY INVESTIGATION, 2005, 85 : 311A - 312A
  • [8] Frequent inactivation of p16(INK4a) in oral premalignant lesions
    Papadimitrakopoulou, V
    Izzo, J
    Lippman, SM
    Lee, JS
    Fan, YH
    Clayman, G
    Ro, JY
    Hittelman, WN
    Lotan, R
    Hong, WK
    Mao, L
    ONCOGENE, 1997, 14 (15) : 1799 - 1803
  • [9] p16(INK4A) gene homozygous deletions in human acute leukaemias with alterations of chromosome 9
    Faienza, MF
    dellaRagione, F
    Basso, G
    Coppola, B
    delGiudice, EM
    Schettini, F
    Iolascon, A
    BRITISH JOURNAL OF HAEMATOLOGY, 1996, 93 (03) : 632 - 636
  • [10] Preferential inactivation of p15(INK4B) but not p16(INK4A) by hypermethylation in T cell acute lymphoblastic leukemia.
    Yu, AL
    Batova, A
    Diccianni, MB
    Pullen, J
    Link, MP
    Yu, J
    BLOOD, 1996, 88 (10) : 1408 - 1408
12345