When the Gut Gets Tough, the Enterocytes Get Going

被引：0
|
作者：
Chen, Grischa Y. [1 ]
Ayres, Janelle S. [1 ]
机构：
[1] Salk Inst Biol Studies, Nomis Ctr Immunobiol & Microbial Pathogenesis, 10010 North Torrey Pines Rd, La Jolla, CA 92103 USA
来源：
IMMUNITY | 2018年 / 48卷 / 05期
关键词：
DROSOPHILA; HOMEOSTASIS; SALMONELLA; MICROBIOTA; TOLERANCE; EXPULSION;
D O I：
10.1016/j.immuni.2018.04.036
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
It is assumed that collateral damage from the immune system drives intestinal epithelial cell (IEC) expulsion during enteric infections. In this issue of Immunity, Zhai et al. (2018) describe how Drosophila's canonical immune deficiency (Imd) pathway programs IEC delamination in the gut.
引用
收藏
页码：837 / 839
页数：3
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