Physiological aspects of low-density lipoprotein oxidation

被引：20
|
作者：
Thomas, MJ [1 ]
机构：
[1] Wake Forest Univ, Bowman Gray Sch Med, Dept Biochem, Winston Salem, NC 27157 USA
来源：
CURRENT OPINION IN LIPIDOLOGY | 2000年 / 11卷 / 03期
关键词：
D O I：
10.1097/00041433-200006000-00011
中图分类号：
Q5 [生物化学]; Q7 [分子生物学];
学科分类号：
071010 ; 081704 ;
摘要：
The oxidation of LDL is thought to be a major contributor to the development of atherosclerosis, Considerable descriptive evidence has been accumulated showing that oxidized LDL promotes pro-atherogenic events. However, direct evidence that oxidized LDL causes atherosclerosis is lacking. This article summarizes the results of recent studies that demonstrate how oxidized LDL affects cellular function, and highlights key issues that should be addressed to link LDL oxidation with atherosclerosis. Curr Opin Lipidol 11:297-301. (C) 2000 Lippincott Williams & Wilkins.
引用
收藏
页码：297 / 301
页数：5
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