Mycobacterium tuberculosis main immune response evasion mechanisms.

被引：0
|
作者：
Chavez-Galan, Leslie [2 ]
del Carmen Arenas-Del Angel, Maria [2 ]
Sada-Ovalle, Isabel [2 ]
Lascurain, Ricardo [1 ]
机构：
[1] Univ Nacl Autonoma Mexico, Dept Bioquim, Fac Med, Mexico City 04510, DF, Mexico
[2] Inst Nacl Enfermedades Resp, Unidad Invest, Dept Bioquim, Mexico City, DF, Mexico
来源：
GACETA MEDICA DE MEXICO | 2009年 / 145卷 / 04期
关键词：
Mycobacterium tuberculosis; avoid immune response; tuberculosis; NITRIC-OXIDE SYNTHASE; INHIBITS MACROPHAGE RESPONSES; CD8; T-CELLS; PHAGOSOME MATURATION; IFN-GAMMA; DENDRITIC CELLS; CLASS-II; UP-REGULATION; APOPTOSIS; PROTEIN;
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
Pulmonary tuberculosis is currently considered a serious health problem worldwide. To understand tuberculosis infection we need to know the interaction between the host's immune response and the microorganism causing tuberculosis. Ample research has been carried out to identify new molecules and genes involved in the evasion mechanism of the host's immune response. This knowledge has generated the possibility that future therapeutic schemes will be designed to fight these particular molecules or genes. We reviewed recent experimental evidence on the mechanisms that will successfully fight against mycobacteria.
引用
收藏
页码：323 / 330
页数：8
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