Activity and safety of CTLA-4 blockade combined with vaccines in cynomolgus macaques

被引:104
|
作者
Keler, T
Halk, E
Vitale, L
O'Neill, T
Blanset, D
Lee, S
Srinivasan, M
Graziano, RF
Davis, T
Lonberg, N
Korman, A
机构
[1] Medarex, Dept Preclin Dev, Bloomsbury, NJ 08804 USA
[2] Medarex, Dept Clin Sci, Bloomsbury, NJ 08804 USA
[3] Medarex, Dept Res & Dev, Milpitas, CA 95035 USA
来源
JOURNAL OF IMMUNOLOGY | 2003年 / 171卷 / 11期
关键词
D O I
10.4049/jimmunol.171.11.6251
中图分类号
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号
100102 ;
摘要
The immune modulatory molecule CTLA-4 (CD152), through interactions with the B7 costimulatory molecules, has been shown to be a negative regulator of T cell activation in various murine model systems. Abs that block CTLA-4 function can enhance immune responses that mediate potent antitumor activity. However, CTLA-4 blockade can also exacerbate autoimmune disease. The safety and activity of anti-CTLA-4 Abs in primates has not been addressed. To that end, we generated human Abs against CTLA-4 using transgenic mice expressing human Ig genes. A high affinity Ab (10D1) that blocked the binding of CTLA-4 to the B7-1 and B7-2 ligands and had cross-reactivity with macaque CTLA-4 was chosen for further development. Administration of 10D1 to cynomolgus macaques significantly enhanced Ab responses to hepatitis surface Ag and a human melanoma cell vaccine. Anti-self Ab responses as measured by immunoassays using lysate from melanocyte-rich tissues were elicited in those animals receiving the melanoma cell vaccine and anti-CTLA-4 Ab. Remarkably, chronic administration of 10D1 did not result in measurable polyclonal T cell activation, significant alteration of the lymphocyte subsets, or induce clinically observable autoimmunity. Repeated dosing of the 10D1 did not elicit monkey anti-human Ab responses in the monkeys. These observations support the development of CTLA-4 blockade for human immunotherapy.
引用
收藏
页码:6251 / 6259
页数:9
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