Attenuation of angiotensin II-induced cardiac hypertrophy in GP91phox-deficient mice.

被引:0
|
作者
Bendall, JK
Heymes, C
Cave, AC
Gall, NP
Shah, AM
机构
[1] Guys Kings & St Thomas Sch Med, London, England
[2] Kings Coll London, GKT Sch Med, London WC2R 2LS, England
关键词
D O I
暂无
中图分类号
R5 [内科学];
学科分类号
1002 ; 100201 ;
摘要
1476
引用
收藏
页码:307 / 307
页数:1
相关论文
共 50 条
  • [1] Angiotensin II-dependent chronic hypertension and cardiac remodeling are independent of tissue superoxide content in gp91phox-deficient mice.
    Reudelhuber, TL
    Mercure, C
    He, Y
    Touyz, RM
    [J]. HYPERTENSION, 2004, 44 (04) : 501 - 502
  • [2] Pivotal role of a gp91phox-containing NADPH oxidase in angiotensin II-induced cardiac hypertrophy in mice
    Bendall, JK
    Cave, AC
    Heymes, C
    Gall, N
    Shah, AM
    [J]. CIRCULATION, 2002, 105 (03) : 293 - 296
  • [3] Statins attenuate angiotensin II-Induced cardiac hypertrophy through inhibition of gp91PHOX-Containing NADPH oxidase
    Nakagami, H
    Liao, JK
    [J]. CIRCULATION, 2003, 108 (17) : 45 - 45
  • [4] A gp91-phox-containing NADPH oxidase is critical for angiotensin II-induced interstitial cardiac fibrosis
    Johar, S
    Cave, AC
    Grieve, DJ
    Shah, AM
    [J]. HEART, 2004, 90 (12)
  • [5] Lipopolysaccharide-induced radical formation in the striatum is abolished in Nox2 gp91phox-deficient mice
    Clement, Hans-Willi
    Vazquez, Juan F.
    Sommer, Olaf
    Heiser, Philip
    Morawietz, Henning
    Hopt, Ulrich
    Schulz, Eberhard
    von Dobschuetz, Ernst
    [J]. JOURNAL OF NEURAL TRANSMISSION, 2010, 117 (01) : 13 - 22
  • [6] Lipopolysaccharide-induced radical formation in the striatum is abolished in Nox2 gp91phox-deficient mice
    Hans-Willi Clement
    Juan F. Vazquez
    Olaf Sommer
    Philip Heiser
    Henning Morawietz
    Ulrich Hopt
    Eberhard Schulz
    Ernst von Dobschütz
    [J]. Journal of Neural Transmission, 2010, 117 : 13 - 22
  • [7] Contrasting mechanisms underlie the development of pressure overload versus angiotensin II-induced hypertrophy in mice lacking the NADPH oxidase subunit gp91phox
    Byrne, JA
    Grieve, DJ
    Bendall, JK
    Cave, AC
    Shah, AM
    [J]. JOURNAL OF PHYSIOLOGY-LONDON, 2002, 544 : 42P - 42P
  • [8] Metabolomics in Angiotensin II-Induced Cardiac Hypertrophy
    Mervaala, Eero
    Biala, Agnieszka
    Merasto, Saara
    Lempiainen, Juha
    Mattila, Ismo
    Martonen, Essi
    Eriksson, Ove
    Louhelainen, Marjut
    Finckenberg, Piet
    Kaheinen, Petri
    Muller, Dominik N.
    Luft, Friedrich C.
    Lapatto, Risto
    Oresic, Matej
    [J]. HYPERTENSION, 2010, 55 (02) : 508 - 515
  • [9] Contrasting mechanisms underlie development of pressure-overload versus angiotensin II-induced hypertrophy in mice lacking the NADPH oxidase subunit gp91phox
    Byrne, JA
    Grieve, DJ
    Bendall, JK
    Cave, A
    Shah, A
    [J]. CIRCULATION, 2002, 106 (19) : 163 - 164
  • [10] Upregulated expression of NADPH oxidase component gp91phox in angiotensin II-infused mice.
    Cifuentes, ME
    Alfie, M
    Carretero, OA
    Pagano, PJ
    [J]. HYPERTENSION, 1999, 34 (02) : 341 - 341