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- [1] Bcl-XL overexpression blocks bax-mediated mitochondrial contact site formation and apoptosis in rod photoreceptors of lead-exposed micePROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2003, 100 (03) : 1022 - 1027He, LHPerkins, GAPoblenz, ATHarris, JBHung, MEllisman, MHFox, DA
- [2] Bcl-xL overexpression in photoreceptors against lead-induced rod-selective mitochondrial dysfunction and apoptosis.INVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE, 2000, 41 (04) : S332 - S332Fox, DAPoblenz, ATHe, LHung, M
- [3] Bcl-xL overexpression blocks bax-mediated mitochondrial contact site formation and apoptosis in rod photoreceptors of lead-exposed mice (vol 100, pg 1022, 2003)PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, 2008, 105 (11) : 4530 - 4530He, LihuaPerkins, Guy A.Poblenz, Ann T.Harris, Jeffrey B.Hung, MichaelEllisman, Mark H.Fox, Donald A.
- [4] Bcl-XL and calyculin A prevent translocation of Bax to mitochondria during apoptosisBIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS, 2002, 291 (05) : 1258 - 1264Ganju, NEastman, A
- [5] Bcl-xL inhibits lead and calcium-induced rod apoptosis by blocking the increase in mitochondrial contact sitesINVESTIGATIVE OPHTHALMOLOGY & VISUAL SCIENCE, 2002, 43 : U433 - U433Fox, DAPerkins, GAHe, LPoblenz, ATEllisman, MHHarris, JB
- [6] The association of deamidation of Bcl-xL and translocation of Bax to the mitochondria through activation of JNK in the induction of apoptosis by treatment with GSH-conjugated DXRINTERNATIONAL JOURNAL OF ONCOLOGY, 2008, 33 (02) : 389 - 395Asakura, TadashiMaeda, KazuhiroOmi, HirokoMatsudaira, HiroshiOhkawa, Kiyoshi