The UV wavelength-specific role of p53 in transcription-coupled nucleotide excision repair is not through differential binding of the p53 protein to its consensus sequence of downstream effectors such as p2l, GADD45, XPC and p48-XPE genes

被引:0
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作者
Dridi, W
Fetni, R
Drobetsky, EA
Drouin, R
机构
[1] Univ Laval, Hosp St Francois, Quebec City, PQ, Canada
[2] Montreal Childrens Hosp, Montreal, PQ H3H 1P3, Canada
[3] Hop Maison Neuve Rosemont, Guy Bernier Res Ctr, Montreal, PQ H1T 2M4, Canada
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暂无
中图分类号
Q3 [遗传学];
学科分类号
071007 ; 090102 ;
摘要
1595
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页码:442 / 442
页数:1
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    [J]. SCIENCE, 1995, 270 (5238) : 1004 - 1005
  • [2] DOES THE P53 UP-REGULATED GADD45 PROTEIN HAVE A ROLE IN EXCISION-REPAIR - REPLY
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    [J]. SCIENCE, 1995, 270 (5238) : 1005 - 1006
  • [3] P53 is preferentially recruited to the promoters of growth arrest genes p2l and GADD45 during replicative senescence of normal human fibroblasts
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    [J]. CANCER RESEARCH, 2006, 66 (17) : 8356 - 8360
  • [4] The role of p53 and downstream effectors in the control of nucleotide excision repair rates in UV-irradiated human cells.
    Therrien, JP
    Drouin, R
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    [J]. PHOTOCHEMISTRY AND PHOTOBIOLOGY, 1999, 69 : 32S - 32S