New Regulatory, Signaling Pathways, and Sources of Nitric Oxide

被引：4
|
作者：
Pluta, Ryszard M. [1 ]
机构：
[1] NINDS, Surg Neurol Branch, NIH, Bethesda, MD 20892 USA
来源：
EARLY BRAIN INJURY OR CEREBRAL VASOSPASM, VOL 1: PATHOPHYSIOLOGY | 2011年 / 110卷
关键词：
Hemoglobin; Neuroglobin; Nitric oxide; SAH; Vasospasm; DELAYED CEREBRAL VASOSPASM; CANINE BASILAR ARTERY; SUBARACHNOID HEMORRHAGE; BLOOD-FLOW; ENDOTHELIAL-CELLS; L-ARGININE; SYNTHASE; MECHANISMS; INHIBITOR; NITRATE;
D O I：
10.1007/978-3-7091-0353-1_2
中图分类号：
R74 [神经病学与精神病学];
学科分类号：
摘要：
Discovered in 1980 by the late Robert F. Furchgott, endothelium-derived relaxing factor, nitric oxide (NO), has been in the forefront of vascular research for several decades. What was originally a narrow approach, has been significantly widened due to major advances in understanding the chemical and biological properties of NO as well as its signaling pathways and discovering new sources of this notorious free radical gas. In this review, recent discoveries regarding NO and their implications on therapy for delayed cerebral vasospasm are presented.
引用
收藏
页码：7 / 12
页数：6
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