Metabolic Encephalopathies

被引：27
|
作者：
Angel, Michael J. [1 ]
Young, G. Bryan [2 ]
机构：
[1] Univ Toronto, Dept Med, Div Neurol, Toronto, ON, Canada
[2] Univ Western Ontario, Div Neurol, Dept Clin Neurol Sci, London Hlth Sci Ctr, London, ON N6A 5A5, Canada
来源：
NEUROLOGIC CLINICS | 2011年 / 29卷 / 04期
关键词：
Metabolic encephalopathy; Altered consciousness; Neurodegeneration; Neuroimaging; REVERSIBLE POSTERIOR LEUKOENCEPHALOPATHY; HYPOGLYCEMIC BRAIN-DAMAGE; DIFFUSION-WEIGHTED MRI; HYPERGLYCEMIC HYPEROSMOLAR STATE; BENZODIAZEPINE RECEPTOR LIGANDS; OBSTRUCTIVE PULMONARY-DISEASE; ELECTRON-MICROSCOPIC FINDINGS; CARBON-MONOXIDE INTOXICATION; IDIOPATHIC RECURRING STUPOR; ACUTE RESPIRATORY-FAILURE;
D O I：
10.1016/j.ncl.2011.08.002
中图分类号：
R74 [神经病学与精神病学];
学科分类号：
摘要：
Kinnier Wilson coined the term metabolic encephalopathy to describe a clinical state of global cerebral dysfunction induced by systemic stress that can vary in clinical presentation from mild executive dysfunction to deep coma with decerebrate posturing; the causes are numerous. Some mechanisms by which cerebral dysfunction occurs in metabolic encephalopathies include focal or global cerebral edema, alterations in transmitter function, the accumulation of uncleared toxic metabolites, postcapillary venule vasogenic edema, and energy failure. This article focuses on common causes of metabolic encephalopathy, and reviews common causes, clinical presentations and, where relevant, management.
引用
收藏
页码：837 / +
页数：47
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