Diminished culpability in fetal alcohol spectrum disorders (FASD)

被引:6
|
作者
Brown, Natalie Novick [1 ]
Greenspan, Stephen [2 ]
机构
[1] Univ Washington, Dept Psychiat & Behav Sci, Seattle, WA 98195 USA
[2] Univ Connecticut, Educ Psychol, Storrs, CT USA
关键词
culpability; executive dysfunction; FASD; fetal alcohol spectrum disorder; ID-equivalence; intellectual disability; EXECUTIVE FUNCTION DEFICITS; INTERROGATIVE SUGGESTIBILITY; CHILDREN; EXPOSURE; ADOLESCENTS; PEOPLE; INDIVIDUALS; ADULTS; DISABILITIES; PREVALENCE;
D O I
10.1002/bsl.2535
中图分类号
B849 [应用心理学];
学科分类号
040203 ;
摘要
There is a biological basis for diminished criminal responsibility in offenders with fetal alcohol spectrum disorders (FASD) just as there is in those with intellectual disability. Functional limitations affecting cognition in both neurodevelopmental conditions stem directly from structural brain damage at a gross and molecular level, which usually impairs executive functioning among other cognitive skills. Executive functioning, which includes reasoning and impulse control, is the only neural system in the brain that involves conscious thought. With respect to the law, impaired reasoning or rationality is an aspect of mens rea ("guilty mind"). When rationality is impaired by prenatal alcohol exposure, acts driven by strong emotion and urges can occur, which has obvious implications regarding criminal responsibility. The Atkins decision by the U.S. Supreme Court reflects the rationale that organically based brain dysfunction in executive skills reduces criminal culpability. We argue that people with FASD who have similar brain dysfunction likewise have reduced criminal responsibility.
引用
收藏
页码:1 / 13
页数:13
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