An imbalance between reactive oxygen species and NO causes endothelial dysfunction and elevated blood pressure in Mas knockout mice

被引：0
|
作者：
Xu, P. [1 ]
Goncalves, A.
Todiras, M. [1 ]
Rabelo, L. [2 ]
Sampaio, W. [3 ]
Moura, M. [3 ]
Santos, S. [3 ]
Luft, F. C. [4 ]
Santos, R. [3 ]
Gross, V [1 ]
Bader, M. [1 ]
Alenina, N. [1 ]
机构：
[1] Max Delbruck Ctr Mol Med, Berlin, Germany
[2] Univ Fed Alagoas, Alagoas, Brazil
[3] Univ Fed Minas Gerais, Belo Horizonte, MG, Brazil
[4] Med Fac Charite, Franz Volhard Clin, Berlin, Germany
来源：
DEUTSCHE MEDIZINISCHE WOCHENSCHRIFT | 2007年 / 132卷 / 46期
关键词：
D O I：
暂无
中图分类号：
R5 [内科学];
学科分类号：
1002 ; 100201 ;
摘要：
引用
收藏
页码：S7 / S7
页数：1
相关论文
共 50 条
  • [1] Endothelial dysfunction and elevated blood pressure in Mas gene-deleted mice
    Xu, Ping
    Costa-Goncalves, Andrey C.
    Todiras, Mihail
    Rabelo, Luiza A.
    Sampaio, Walkyria O.
    Moura, Marina M.
    Santos, Sergio Sousa
    Luft, Friedrich C.
    Bader, Michael
    Gross, Volkmar
    Alenina, Natalia
    Santos, Robson A. S.
    HYPERTENSION, 2008, 51 (02) : 574 - 580
  • [2] The accelerated endothelial dysfunction associated with insulin resistance is mediated by reactive oxygen species - Studies in mice heterozygous for a knockout of the insulin receptor
    Duncan, E
    Wheatcroft, S
    Ezzat, V
    Li, JM
    Shah, A
    Kearney, M
    CIRCULATION, 2005, 112 (17) : U368 - U368
  • [3] Insulin resistance causes premature endothelial dysfunction through increased production of reactive oxygen species
    Duncan, E. R.
    Walker, S. J.
    Ezzat, V. A.
    Wheatcroft, S. B.
    Li, J. . M.
    Shah, A. M.
    Kearney, M. T.
    ATHEROSCLEROSIS, 2006, 186 (02) : S5 - S5
  • [4] Coronary endothelial dysfunction and mitochondrial reactive oxygen species in type 2 diabetic mice
    Cho, Young-Eun
    Basu, Aninda
    Dai, Anzhi
    Heldak, Michael
    Makino, Ayako
    AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY, 2013, 305 (10): : C1033 - C1040
  • [5] Reactive Oxygen and Nitrogen Species Impact on Endothelial Dysfunction
    Salisbury, Dereck
    Bronas, Ulf
    NURSING RESEARCH, 2015, 64 (01) : 53 - 66
  • [6] DELETION OF RECEPTER ATTENUATES VASCULAR ENDOTHELIAL DYSFUNCTION AND OXYGEN SPECIES PRODUCTION IN LDL RECEPTOR KNOCKOUT MICE
    Sumiyoshi, K.
    Shimada, K.
    Iesaki, T.
    Miyazaki, T.
    Kume, A.
    Kiyanagi, T.
    Hiki, M.
    Fukao, K.
    Hirose, K.
    Matsumori, R.
    Tada, N.
    Mokuno, H.
    Daida, H.
    ATHEROSCLEROSIS SUPPLEMENTS, 2008, 9 (01) : 65 - 65
  • [7] Reactive oxygen species are involved in baseline arterial blood pressure and L-NAME-induced hypertension in mas-deficient mice
    Costa-Goncalves, Andrey C.
    Tank, Jens
    Plehm, Ralph
    Bader, Michael
    Alenina, Natalia
    Santos, Robson A.
    Jordan, Jens
    Luft, Friedrich C.
    Gross, Volkmar
    HYPERTENSION, 2007, 50 (04) : E126 - E126
  • [8] TNF-a induced eNOS uncoupling mediates endothelial dysfunction through elevated reactive oxygen species
    Farrar, Emily Jean
    Huntley, Geoffrey
    Hsu, Kevin S.
    Butcher, Jonathan T.
    FASEB JOURNAL, 2013, 27
  • [9] Lipoteichoic acid (LTA) causes reactive oxygen species (ROS) dependent lung endothelial barrier dysfunction
    Johnson, A.
    Barton-Pai, A.
    AMERICAN JOURNAL OF RESPIRATORY AND CRITICAL CARE MEDICINE, 2010, 181
  • [10] Endothelial dysfunction driven by mitochondrial reactive oxygen species - proof of concept studies in CypD -/- mice
    Daub, Steffen
    Kroeller-Schoen, Swenja
    Steven, Sebastian
    Kossmann, Sabine
    Scholz, Alexander
    Oelze, Matthias
    Xia, Ning
    Treiber, Nicolai
    Scharffetter-Kochanek, Karin
    Li, Huige
    Schulz, Eberhard
    Wenzel, Philip
    Muenzel, Thomas
    Daiber, Andreas
    FASEB JOURNAL, 2013, 27
12345