Antibacterial cathelicidin peptide human CAP18/LL-37 suppresses neutrophil apoptosis via the activation of FPRL1 and P2X7

被引：0
|
作者：
Nagaoka, I
Tsutsumi-Ishii, Y
Tamura, H
Hirata, M
机构：
[1] Juntendo Univ, Tokyo 1138421, Japan
[2] Seikagaku Corp, Tokyo, Japan
[3] Ohtaka Enzyne Co, Hokkaido, Japan
来源：
JOURNAL OF LEUKOCYTE BIOLOGY | 2005年
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D O I：
暂无
中图分类号：
Q2 [细胞生物学];
学科分类号：
071009 ; 090102 ;
摘要：
186
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页码：74 / 75
页数：2
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  • [3] Effect of antibacterial cathelicidin peptide CAP18/LL-37 on sepsis in neonatal rats
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  • [5] The Human Cathelicidin LL-37 Modulates the Activities of the P2X7 Receptor in a Structure-dependent Manner
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    [J]. JOURNAL OF BIOLOGICAL CHEMISTRY, 2008, 283 (45) : 30471 - 30481
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    [J]. JOURNAL OF INVESTIGATIVE DERMATOLOGY, 2023, 143 (05) : 751 - 761.e7
  • [7] LL-37, the neutrophil granule- and epithelial cell-derived cathelicidin, utilizes formyl peptide receptor-like 1 (FPRL1) as a receptor to chemoattract human peripheral blood neutrophils, monocytes, and T cells
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