Celiac disease: a model disease for gene-environment interaction

被引：7
|
作者：
Uibo, Raivo [2 ]
Tian, Zhigang [3 ]
Gershwin, M. Eric [1 ]
机构：
[1] Univ Calif Davis, Div Rheumatol Allergy & Clin Immunol, Sch Med, Davis, CA 95616 USA
[2] Univ Tartu, Immunol Grp, IGMP, EE-50090 Tartu, Estonia
[3] Univ Sci & Technol China, Hefei 230026, Peoples R China
来源：
CELLULAR & MOLECULAR IMMUNOLOGY | 2011年 / 8卷 / 02期
关键词：
environment and autoimmunity; geoepidemiology; autoimmunity; INTESTINAL PERMEABILITY; GEOEPIDEMIOLOGY; AUTOIMMUNITY; AUTOANTIBODIES; EPIDEMIOLOGY; INFLAMMATION; CHILDHOOD; SCLEROSIS; RESPONSES;
D O I：
10.1038/cmi.2010.62
中图分类号：
R392 [医学免疫学]; Q939.91 [免疫学];
学科分类号：
100102 ;
摘要：
Celiac sprue remains a model autoimmune disease for dissection of genetic and environmental influences on disease progression. The 2010 Congress of Autoimmunity included several key sessions devoted to genetics and environment. Several papers from these symposia were selected for in-depth discussion and publication. This issue is devoted to this theme. The goal is not to discuss genetic and environmental interactions, but rather to focus on key elements of diagnosis, the inflammatory response and the mechanisms of autoimmunity. Cellular & Molecular Immunology (2011) 8, 93-95; doi: 10.1038/cmi.2010.62; published online 14 February 2011
引用
收藏
页码：93 / 95
页数：3
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